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The document outlines the progression of infections in open wounds and fractures, highlighting predisposing factors such as vascular insufficiency, IV drug use, and immunocompromising diseases. It details how microorganisms enter the body, grow, and lead to symptoms like fever and bone pain, ultimately resulting in complications such as necrosis and the formation of sequestra. The chronic stage of infection can lead to scar tissue development and potential loss of organ function, with amputation as a possible outcome.

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Enzo Dy
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31 views2 pages

5

The document outlines the progression of infections in open wounds and fractures, highlighting predisposing factors such as vascular insufficiency, IV drug use, and immunocompromising diseases. It details how microorganisms enter the body, grow, and lead to symptoms like fever and bone pain, ultimately resulting in complications such as necrosis and the formation of sequestra. The chronic stage of infection can lead to scar tissue development and potential loss of organ function, with amputation as a possible outcome.

Uploaded by

Enzo Dy
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOCX, PDF, TXT or read online on Scribd
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Open wounds/

Predisposing factors: fractures

vascular insufficiency disorders


genitourinary infections Microorganisms gain
respiratory infections entry
IV drug use by way of blood
immunocompromising diseases
history of blood- stream
infections Microorganisms lodge into
Indwelling prosthetic devices an area where circulation
slows

Microorganisms grow

Increase pressure

Vascular compromise fever, night sweats,


Ischemia of the periosteum chills, restlessness,
nausea and malaise
constant bone pain,
Infection through the bone swelling, tenderness,
cortex and marrow warmth at the infection site,
restricted movement
Cortical devascularization of the affected part

Debridement necrosis

Formation of new bone Separation of devitalized


Sequestra
bone from living bone

Involcrum
Continues to be an
infected island

Difficulty to reach by
blood borne antibiotics

Chronic Enlarged sequestrum


stage

drainage from
sinus tracts Development of sinus tract Sequestrum move
out to the soft tissue

Turns to scar tissue


revascularized
Systemic signs may
be diminished with Site for continued
Removal by the normal
constant bone pain, microorganism growth
immune process
Swelling, tenderness,
warmth at the infection
site Remission and
exacerbation Healing

Excessive vascular
insufficiency

Loss of organ
function
Amputation

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