How to Read a CTG
geekymedics.com/how-to-read-a-ctg/
Dr Lewis Potter March 29, 2011
What is cardiotocography?
Cardiotocography (CTG) is used during pregnancy to monitor fetal heart rate and
uterine contractions. It is most commonly used in the third trimester and its purpose is to
monitor fetal well-being and allow early detection of fetal distress. An abnormal CTG may
indicate the need for further investigations and potential intervention.
Check out our CTG quiz on the Geeky Medics quiz platform to put your CTG
interpretation knowledge to the test.
How CTG works
The device used in cardiotocography is known as a cardiotocograph. It involves the
placement of two transducers onto the abdomen of a pregnant woman. One transducer
records the fetal heart rate using ultrasound and the other transducer monitors the
contractions of the uterus by measuring the tension of the maternal abdominal wall
(providing an indirect indication of intrauterine pressure). The CTG is then assessed by
a midwife and the obstetric medical team.
How to read a CTG
To interpret a CTG you need a structured method of assessing its various characteristics.
The most popular structure can be remembered using the acronym DR C BRAVADO:
DR: Define risk
C: Contractions
BRa: Baseline rate
V: Variability
A: Accelerations
D: Decelerations
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O: Overall impression
You might also be interested in our OSCE Flashcard Collection which contains over
2000 flashcards that cover clinical examination, procedures, communication skills
and data interpretation.
Define risk
When performing CTG interpretation, you first need to determine if the pregnancy is high
or low risk. This is important as it gives more context to the CTG reading (e.g. if the
pregnancy categorised as high-risk, the threshold for intervention may be lower). Some
reasons a pregnancy may be considered high risk are shown below.1
Maternal medical illness
Gestational diabetes
Hypertension
Asthma
Obstetric complications
Multiple gestation
Post-date gestation
Previous cesarean section
Intrauterine growth restriction
Premature rupture of membranes
Congenital malformations
Oxytocin induction/augmentation of labour
Pre-eclampsia
Other risk factors
Absence of prenatal care
Smoking
Drug abuse
Contractions
Next, you need to record the number of contractions present in a 10 minute period.
Each big square on the example CTG chart below is equal to one minute, so look at
how many contractions occurred within 10 big squares.
Individual contractions are seen as peaks on the part of the CTG monitoring uterine
activity.
Assess contractions for the following:
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Duration: How long do the contractions last?
Intensity: How strong are the contractions (assessed using palpation)?
In the below example, there are 2 contractions in a 10 minute period (this is often
referred to as “2 in 10”).
Uterine contractions (CTG)
Baseline rate of the fetal heart
The baseline rate is the average heart rate of the fetus within a 10-minute window.
Look at the CTG and assess what the average heart rate has been over the last 10
minutes, ignoring any accelerations or decelerations.
A normal fetal heart rate is between 110-160 bpm.
CTG: Baseline heart rate
Fetal tachycardia
Fetal tachycardia is defined as a baseline heart rate greater than 160 bpm.
Causes of fetal tachycardia include:
Fetal hypoxia
Chorioamnionitis
Hyperthyroidism
Fetal or maternal anaemia
Fetal tachyarrhythmia
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Fetal bradycardia
Fetal bradycardia is defined as a baseline heart rate of less than 100 bpm.
It is common to have a baseline heart rate of between 100-120 bpm in the following
situations:
Postdate gestation
Occiput posterior or transverse presentations
Severe prolonged bradycardia (less than 80 bpm for more than 3 minutes) indicates
severe hypoxia.
Causes of prolonged severe bradycardia include:
Prolonged cord compression
Cord prolapse
Epidural and spinal anaesthesia
Maternal seizures
Rapid fetal descent
Variability
Baseline variability refers to the variation of fetal heart rate from one beat to the next.
Variability occurs as a result of the interaction between the nervous system,
chemoreceptors, baroreceptors and cardiac responsiveness.
It is, therefore, a good indicator of how healthy a fetus is at that particular moment in time,
as a healthy fetus will constantly be adapting its heart rate in response to changes in its
environment.
Normal variability indicates an intact neurological system in the fetus.
Normal variability is between 5-25 bpm.3
To calculate variability you need to assess how much the peaks and troughs of the heart
rate deviate from the baseline rate (in bpm).
Variability categorisation
Variability can be categorised as either reassuring, non-reassuring or abnormal. 3
Reassuring: 5 – 25 bpm
Non-reassuring:
less than 5 bpm for between 30-50 minutes
more than 25 bpm for 15-25 minutes
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Abnormal:
less than 5 bpm for more than 50 minutes
more than 25 bpm for more than 25 minutes
sinusoidal
CTG: Variability
Reduced variability can be caused by any of the following:2
Fetal sleeping: this should last no longer than 40 minutes (this is the most common
cause)
Fetal acidosis (due to hypoxia): more likely if late decelerations are also present
Fetal tachycardia
Drugs: opiates, benzodiazepines, methyldopa and magnesium sulphate
Prematurity: variability is reduced at earlier gestation (<28 weeks)
Congenital heart abnormalities
CTG: Reduced variability
Accelerations
Accelerations are an abrupt increase in the baseline fetal heart rate of greater than
15 bpm for greater than 15 seconds.1
The presence of accelerations is reassuring.
Accelerations occurring alongside uterine contractions is a sign of a healthy fetus.
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The absence of accelerations with an otherwise normal CTG is of uncertain
significance.
CTG: Accelerations
Decelerations
Decelerations are an abrupt decrease in the baseline fetal heart rate of greater than
15 bpm for greater than 15 seconds.
The fetal heart rate is controlled by the autonomic and somatic nervous system. In
response to hypoxic stress, the fetus reduces its heart rate to preserve myocardial
oxygenation and perfusion. Unlike an adult, a fetus cannot increase its respiration depth
and rate. This reduction in heart rate to reduce myocardial demand is referred to as a
deceleration.
There are a number of different types of decelerations, each with varying significance.
Early deceleration
Early decelerations start when the uterine contraction begins and recover when
uterine contraction stops. This is due to increased fetal intracranial pressure causing
increased vagal tone. It therefore quickly resolves once the uterine contraction ends and
intracranial pressure reduces. This type of deceleration is, therefore, considered to be
physiological and not pathological.3
CTG: Early decelerations
Variable deceleration
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Variable decelerations are observed as a rapid fall in baseline fetal heart rate with a
variable recovery phase.
They are variable in their duration and may not have any relationship to uterine
contractions.
They are most often seen during labour and in patients’ with reduced amniotic fluid
volume.
All fetuses experience stress during the labour process, as a result of uterine contractions
reducing fetal perfusion. Whilst fetal stress is to be expected during labour, the challenge
is to pick up pathological fetal distress.
Variable decelerations are usually caused by umbilical cord compression. The
mechanism is as follows:1
1. The umbilical vein is often occluded first causing an acceleration of the fetal heart rate
in response.
2. Then the umbilical artery is occluded causing a subsequent rapid deceleration.
3. When pressure on the cord is reduced another acceleration occurs and then the
baseline rate returns.
The accelerations before and after a variable deceleration are known as the
shoulders of deceleration. Their presence indicates the fetus is not yet hypoxic and is
adapting to the reduced blood flow. Variable decelerations can sometimes resolve if the
mother changes position. The presence of persistent variable decelerations indicates the
need for close monitoring. Variable decelerations without the shoulders are more
worrying, as it suggests the fetus is becoming hypoxic.
CTG: Variable decelerations
Late deceleration
Late decelerations begin at the peak of the uterine contraction and recover after the
contraction ends. This type of deceleration indicates there is insufficient blood flow to
the uterus and placenta. As a result, blood flow to the fetus is significantly reduced
causing fetal hypoxia and acidosis.
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Causes of reduced uteroplacental blood flow include:1
Maternal hypotension
Pre-eclampsia
Uterine hyperstimulation
CTG: Late decelerations
Prolonged deceleration
A prolonged deceleration is defined as a deceleration that lasts more than 2 minutes:
If it lasts between 2-3 minutes it is classed as non-reassuring.
If it lasts longer than 3 minutes it is immediately classed as abnormal.
CTG: Prolonged deceleration
Sinusoidal pattern
A sinusoidal CTG pattern is rare, however, if present it is very concerning as it is
associated with high rates of fetal morbidity and mortality.1
A sinusoidal CTG pattern has the following characteristics:
A smooth, regular, wave-like pattern
Frequency of around 2-5 cycles a minute
Stable baseline rate around 120-160bpm
No beat to beat variability
A sinusoidal pattern usually indicates one or more of the following:
Severe fetal hypoxia
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Severe fetal anaemia
Fetal/maternal haemorrhage
CTG: Sinusoidal pattern
Overall impression
Once you have assessed all aspects of the CTG you need to determine your overall
impression.
The overall impression can be described as either reassuring, suspicious or
abnormal.3
Overall impression is determined by how many of the CTG features were either
reassuring, non-reassuring or abnormal. The NICE guidelines below demonstrate how to
decide which category a CTG falls into.3
Reassuring
Baseline heart rate
110 to 160 bpm
Baseline variability
5 to 25 bpm
Decelerations
None or early
Variable decelerations with no concerning characteristics for less than 90 minutes
Non-reassuring
Baseline heart rate
Either of the below would be classed as non-reassuring:
100 to 109 bpm
161 to 180 bpm
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Baseline variability
Either of the below would be classed as non-reassuring:
Less than 5 for 30 to 50 minutes
More than 25 for 15 to 25 minutes
Decelerations
Any of the below would be classed as non-reassuring:
Variable decelerations with no concerning characteristics for 90 minutes or more.
Variable decelerations with any concerning characteristics in up to 50% of
contractions for 30 minutes or more.
Variable decelerations with any concerning characteristics in over 50% of
contractions for less than 30 minutes.
Late decelerations in over 50% of contractions for less than 30 minutes, with no
maternal or fetal clinical risk factors such as vaginal bleeding or significant
meconium.
Abnormal
Baseline heart rate
Either of the below would be classed as abnormal:
Below 100 bpm
Above 180 bpm
Baseline variability
Any of the below would be classed as abnormal:
Less than 5 for more than 50 minutes
More than 25 for more than 25 minutes
Sinusoidal
Decelerations
Any of the below would be classed as abnormal:
Variable decelerations with any concerning characteristics in over 50% of
contractions for 30 minutes (or less if any maternal or fetal clinical risk factors – see
above).
Late decelerations for 30 minutes (or less if any maternal or fetal clinical risk
factors).
Acute bradycardia, or a single prolonged deceleration lasting 3 minutes or more.
Regard the following as concerning characteristics of variable decelerations:
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Lasting more than 60 seconds
Reduced baseline variability within the deceleration
Failure to return to baseline
Biphasic (W) shape
No shouldering
Reviewer
Dr Venkatesh Subramanian
Obstetrics & Gynaecology Registrar in London
References
1. AMIR SWEHA, M.D. Interpretation of the Electronic Fetal Heart Rate During Labor.
Am Fam Physician. 1999 May 1;59(9):2487-2500. Available from: [LINK].
2. Clinical obstetrics and gynaecology. 2nd Edition. 2009. B.Magowan, Philip Owen,
James Drife.
3. Intrapartum care: NICE guideline CG190 (February 2017). Available from: [LINK].
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