PO No :PO3166792715-286

Name : Ms.POOJA JAIN Client Name : TATA 1MG OKHLA

Age/Gender : 39/Female Registration Date : 03/May/2024 11:22AM
Patient ID : OKH1306391 Collection Date : 03/May/2024 09:33AM
Barcode ID/Order ID : D9107940 / 9520773 Sample Receive Date : 03/May/2024 12:49PM
Referred By : Dr. Report Status : Final Report
Sample Type : Serum Report Date : 03/May/2024 04:14PM

BIOCHEMISTRY
KIDNEY FUNCTION TEST & LIVER FUNCTION TEST
Test Name Result Unit Bio. Ref. Interval Method

Liver Function Test

Bilirubin-Total 0.51 mg/dl 0.3 – 1.2 Vanadate oxidation
Bilirubin-Direct 0.15 mg/dl 0-0.3 Vanadate oxidation
Bilirubin-Indirect 0.36 mg/dL 0.2-0.8 Calculated
Protein, Total 6.60 g/dL 5.7-8.2 Biuret
Albumin 3.28 g/dL 3.4-4.8 BCG Dye Binding
Globulin 3.3 g/dl 2.3 - 4.1 Calculated
A/G Ratio 0.99 Ratio 0.8 - 1.9 Calculated
Aspartate Transaminase (SGOT) 34 U/L <34 Modified IFCC
Alanine Transaminase (SGPT) 48 U/L 10-49 Modified IFCC
SGOT/SGPT 0.71 Ratio <1 Calculated
Alkaline Phosphatase 292 U/L 46-116 IFCC Standardization
Gamma Glutamyltransferase (GGT) 43 U/L <38 Modified IFCC

Comment:
•LFTS are based upon measurements of substances released from damaged hepatic cells into the blood that gives idea of the
Existence, Extent and Type of Liver damage. - Acute Hepatocellular damage: ALT & AST levels are sensitive index of
hepatocellular damage - Obstruction to the biliary tract,Cholestasis and blockage of bile flow:1) Serum Total Bilirubin
concentration 2) Serum Alkaline Phosphatase (ALP) activity 3) Gamma Glutamyl Transpeptidase (GGTP) 4) 5`-Nucleotidase -
Chronic liver disease: Serum Albumin concentration
•Bilirubin results from the enzymatic breakdown of heme. Jaundice is a yellowish discoloration of the skin and mucous
membranes caused by hyperbilirubinemia.
•Pre-hepatic or hemolytic jaundice - Abnormal red cells, antibodies,drugs and toxins,Hemoglobinopathies, Gilbert’s syndrome,
Crigler-Najjar syndrome
•Hepatic or Hepatocellular jaundice-Viral hepatitis,toxic hepatitis, intrahepatic cholestasis
•Post-hepatic jaundice -Extrahepatic cholestasis, gallstones, tumors of the bile duct, carcinoma of pancreas
•In viral hepatitis and other forms of liver disease associated with acute hepatic necrosis, serum AST and ALT concentrations are
elevated even before the clinical signs and symptoms of disease appear.
•ALT is the more liver-specific enzyme and elevations of ALT activity persist longer than AST activity.
•Peak values of aminotransferase activity occur between the seventh and twelfth days. Activities then gradually decrease,
reaching normal activities by the third to fifth week. Peak activities bear no relationship to prognosis and may fall with worsening
of the patient's condition.
•Aminotransferase activities observed in cirrhosis vary with the status of the cirrhotic process and range from the upper
reference limit to four to five times higher, with an AST/ALT ratio greater than 1. The ratio's elevation can reflect the grade of
fibrosis in these patients. Slight or moderate elevations of both AST and ALT activities have been observed after administration
of various medications and chronic hepatic injury such as (1) hemochromatosis, (2) Wilson disease, (3) autoimmune hepatitis, (4)
primary biliary cirrhosis, (5) sclerosing cholangitis, and (6) a1-antitrypsin deficiency.
•AST activity also is increased in acute myocardial infarction, progressive muscular dystrophy and dermatomyositis, reaching
concentrations up to eight times the upper reference limit.Slight to moderate AST elevations are noted in hemolytic disease.
•GGT is a sensitive indicator of the presence of hepatobiliary disease, being elevated in most subjects with liver disease

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TATA 1MG OKHLA
Address: 2nd Floor, B-225, Okhla Phase I,
Okhla Industrial Estate, New Delhi, Delhi 110020

Page 1 of 4
PO No :PO3166792715-286

Name : Ms.POOJA JAIN Client Name : TATA 1MG OKHLA

Age/Gender : 39/Female Registration Date : 03/May/2024 11:22AM
Patient ID : OKH1306391 Collection Date : 03/May/2024 09:33AM
Barcode ID/Order ID : D9107940 / 9520773 Sample Receive Date : 03/May/2024 12:49PM
Referred By : Dr. Report Status : Final Report
Sample Type : Serum Report Date : 03/May/2024 04:14PM

BIOCHEMISTRY
KIDNEY FUNCTION TEST & LIVER FUNCTION TEST
Test Name Result Unit Bio. Ref. Interval Method
regardless of cause. Increased concentrations of the enzyme are also found in serum of subjects receiving anticonvulsant drugs,
such as phenytoin and phenobarbital.

Scan for
This test has been performed at digital copy
TATA 1MG OKHLA
Address: 2nd Floor, B-225, Okhla Phase I,
Okhla Industrial Estate, New Delhi, Delhi 110020

Page 2 of 4
 PO No :PO3166792715-286

Name : Ms.POOJA JAIN Client Name : TATA 1MG OKHLA

Age/Gender : 39/Female Registration Date : 03/May/2024 11:22AM
Patient ID : OKH1306391 Collection Date : 03/May/2024 09:33AM
Barcode ID/Order ID : D9107940 / 9520773 Sample Receive Date : 03/May/2024 12:49PM
Referred By : Dr. Report Status : Final Report
Sample Type : Serum Report Date : 03/May/2024 04:09PM

BIOCHEMISTRY
KIDNEY FUNCTION TEST & LIVER FUNCTION TEST
Test Name Result Unit Bio. Ref. Interval Method

Kidney Function Test.

Blood Urea Nitrogen 10 mg/dL 9-23 Urease with GLDH
Urea 21.72 mg/dl 19.26-49.22 Calculated
Creatinine 0.54 mg/dL 0.5-1.1 Alkaline picrate - kinetic
Uric Acid 5.0 mg/dL 2.7-6.1 Uricase/Peroxidase
Sodium 137 mmol/L 132-146 Indirect ISE
Potassium 4.60 mmol/L 3.5-5.5 Indirect ISE
Chloride 107.0 mmol/L 99-109 Indirect ISE
BUN/Creatinine Ratio 18.8 Ratio 12:1 - 20:1 Calculated

Comment:
BUN is directly related to protein intake and nitrogen metabolism and inversely related to the rate of excretion of urea.Blood
urea nitrogen (BUN) levels reflect the balance between the production and excretion of urea. Increased levels are seen in renal
failure (acute or chronic), urinary tract obstruction, dehydration, shock, burns, CHF, GI bleeding, nephrotoxic drugs. Decreased
levels are seen in hepatic failure, nephrotic syndrome, cachexia (low-protein and high-carbohydrate diets).
Urea is a non-proteinous nitrogen compound formed in the liver from ammonia as an end product of protein metabolism. Urea
diffuses freely into extracellular and intracellular fluid and is ultimately excreted by the kidneys. Increased levels are found in
acute renal failure, chronic glomerulonephritis, congestive heart failure, decreased renal perfusion, diabetes, excessive protein
ingestion, gastrointestinal (GI) bleeding, hyperalimentation, hypovolemia, ketoacidosis, muscle wasting from starvation,
neoplasms, pyelonephritis, shock, urinary tract obstruction, nephrotoxic drugs. Decreased levels are seen in inadequate dietary
protein, low-protein/high-carbohydrate diet, malabsorption syndromes, pregnancy, severe liver disease, certain drugs.
Creatinine is catabolic product of creatinine phosphate, which is excreted by filtration through the glomerulus and by tubular
secretion. Creatinine clearance is an acceptable clinical measure of glomerular filtration rate (GFR). Increased levels are seen in
acute/chronic renal failure, urinary tract obstruction, hypothyroidism, nephrotoxic drugs, shock, dehydration, congestive heart
failure, diabetes. Decreased levels are found in muscular dystrophy.
BUN/Creatinine ratio (normally 12:1–20:1) is decreased in acute tubular necrosis, advanced liver disease, low protein intake,
and following hemodialysis. BUN/Creatinine ratio is increased in dehydration, GI bleeding, and increased catabolism.
Uric acid levels show diurnal variation. The level is usually higher in the morning and lower in the evening. Increased levels are
seen in starvation, strenuous exercise, malnutrition, or lead poisoning, gout, renal disorders, increased breakdown of body cells
in some cancers (including leukemia, lymphoma, and multiple myeloma) or cancer treatments, hemolytic anemia, sickle cell
anemia, or heart failure, pre-eclampsia, liver disease (cirrhosis), obesity, psoriasis, hypothyroidism, low blood levels of
parathyroid hormone (PTH), certain drugs, foods that are very high in purines - such as organ meats, red meats, some seafood
and beer. Decreased levels are seen in liver disease, Wilson's disease, Syndrome of inappropriate antidiuretic hormone (SIADH),
certain drugs.

*** End Of Report ***

Conditions of Laboratory Testing & Reporting:
Test results released pertain to the sample, as received. Laboratory investigations are only a tool to facilitate in arriving at a diagnosis and should
be clinically correlated by the interpreting clinician. Result delays may happen because of unforeseen or uncontrollable circumstances. Test report

Scan for
This test has been performed at digital copy
TATA 1MG OKHLA
Address: 2nd Floor, B-225, Okhla Phase I,
Okhla Industrial Estate, New Delhi, Delhi 110020

Page 3 of 4
 PO No :PO3166792715-286

Name : Ms.POOJA JAIN Client Name : TATA 1MG OKHLA

Age/Gender : 39/Female Registration Date : 03/May/2024 11:22AM
Patient ID : OKH1306391 Collection Date : 03/May/2024 09:33AM
Barcode ID/Order ID : D9107940 / 9520773 Sample Receive Date : 03/May/2024 12:49PM
Referred By : Dr. Report Status : Final Report
Sample Type : Serum Report Date : 03/May/2024 04:09PM

BIOCHEMISTRY
KIDNEY FUNCTION TEST & LIVER FUNCTION TEST
Test Name Result Unit Bio. Ref. Interval Method
may vary depending on the assay method used. Test results may show inter-laboratory variations. Test results are not valid for medico-legal
purposes. Please mail your queries related to test results to Customer Care mall ID care@1mg.com

Disclaimer: Results relate only to the sample received. Test results marked "BOLD" indicate abnormal results i.e. higher or lower than normal. All
lab test results are subject to clinical interpretation by a qualified medical professional. This report cannot be used for any medico-legal purposes.
Partial reproduction of the test results is not permitted. Also, TATA 1mg Labs is not responsible for any misinterpretation or misuse of the
information. The test reports alone may not be conclusive of the disease/condition, hence clinical correlation is necessary. Reports should be
vetted by a qualified doctor only.

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TATA 1MG OKHLA
Address: 2nd Floor, B-225, Okhla Phase I,
Okhla Industrial Estate, New Delhi, Delhi 110020

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