CLIENT DETAILS Medimaa Diagnostics Pvt. Ltd.
CLIENT CODE: MED000015 A-72, Sector-50, Noida, Uttar Pradesh 201301,INDIA
MEDIMAA IMAGING PVT LTD Email: care@medimaa.in
483,RAM SHREE COMPLEX,HOSIYAR Website: www.medimaahealthcare.com
PUR,SECTOR 51 NOIDA,201301 Ph: 0120-4234948
11210022
Mobile: 8527852799 ,Ph: 8527852798
Email:
PATIENT NAME Mrs. MUKHTYARI DEVI PATIENT ID 10257199
LAB NO 11210022 AGE 75 Y GENDER Female DATE OF BIRTH
DRAWN 30/07/2024 16:48 RECEIVED 30/07/2024 16:50 REPORTED 30/07/2024 18:13
REF. BY Dr. ROBZ HOSPITAL Report Status Final
PATIENT HISTORY
Investigations Result Biological Ref.Int. Unit
BIOCHEMISTRY
IRON PROFILE
IRON 28.8 Low WOMEN 50.0-170.0 ug/dL
Method: Pyridium azo dye NEWBORN 100.0-250.0
INFANT 40.0-100.0
CHILD 50.0-120.0
UIBC 226.3 110.0-370.0 ug/dL
Method: BIOCHEMICAL
TOTAL IRON BINDING CAPACITY 255.1 228.0-428.0 ug/dL
Method: SPECTROPHOTOMETRY
TRANSFERRIN SATURATION 11.29 Low 20.0-50.0 %
Method: CALCULATED
*** End Of Report ***
Page 1 of 4
CLIENT DETAILS Medimaa Diagnostics Pvt. Ltd.
CLIENT CODE: MED000015 A-72, Sector-50, Noida, Uttar Pradesh 201301,INDIA
MEDIMAA IMAGING PVT LTD Email: care@medimaa.in
483,RAM SHREE COMPLEX,HOSIYAR Website: www.medimaahealthcare.com
PUR,SECTOR 51 NOIDA,201301 Ph: 0120-4234948
11210022
Mobile: 8527852799 ,Ph: 8527852798
Email:
PATIENT NAME Mrs. MUKHTYARI DEVI PATIENT ID 10257199
LAB NO 11210022 AGE 75 Y GENDER Female DATE OF BIRTH
DRAWN 30/07/2024 16:48 RECEIVED 30/07/2024 16:50 REPORTED 30/07/2024 18:13
REF. BY Dr. ROBZ HOSPITAL Report Status Final
PATIENT HISTORY
Investigations Result Biological Ref.Int. Unit
IMMUNOLOGY
IMMUNOLOGY
VITAMIN B12,SERUM
VITAMIN B12 LEVEL ,SERUM 1479.6 High 200.0-1100.0 pg/mL
Method: CLIA
Interpretation
Vitamin B12 belongs to the corrin family it is a cofactor for the conversion of methylemalonyl coenzyme A to succinyl coenzyme A, synthesis of methionine
from homocysteine and formation of myelin. It is required along with folate for DNA synthesis. The major source of vitamin B12 for human beings is meat, while
herbivorous animals get their requirement from contaminated vegetable matter and coprophagy. Megaloblastic anaemia can be due to cobalamin & or folic acid
deficiency.
Vitamin B12 or Cyanocobalamin, is a complex corrinoid compound containing four pyrrole rings that surround a single cobalt atom. Humans obtain vitamin B12
exclusively from animal dietary sources,such as meat, eggs and milk.
Clinical and laboratory findings for Vitamin B12 deficiency include neurological abnormalities,decreased serum B12 levels and increased excretion of
methylmalonic acid. The impaired DNA synthesis associated with Vitamin B12 deficiency causes macrocytic anaemias. These anaemias are characterized by
abnormal maturation of erythrocyte precursors in the Bone-Marrow, which results in the presence of magaloblasts and in decreased erythrocyte survival.
Pernicious anaemia is a macrocytic anaemia caused by Vitamin B12 deficiency that is due to lack of intrinsic factor. Low Vitamin B12 intake, gastrectomy,
diseases of small intestine, malabsorption and trans-cobalamin deficiency can also cause Vitamin B12 deficiency.
Increased Levels :-1. Renal failure 2. Liver disease
Decreased Levels :-1. Megaloblastic anemia 2. Vegetarianism
*** End Of Report ***
Page 2 of 4
CLIENT DETAILS Medimaa Diagnostics Pvt. Ltd.
CLIENT CODE: MED000015 A-72, Sector-50, Noida, Uttar Pradesh 201301,INDIA
MEDIMAA IMAGING PVT LTD Email: care@medimaa.in
483,RAM SHREE COMPLEX,HOSIYAR Website: www.medimaahealthcare.com
PUR,SECTOR 51 NOIDA,201301 Ph: 0120-4234948
11210022
Mobile: 8527852799 ,Ph: 8527852798
Email:
PATIENT NAME Mrs. MUKHTYARI DEVI PATIENT ID 10257199
LAB NO 11210022 AGE 75 Y GENDER Female DATE OF BIRTH
DRAWN 30/07/2024 16:48 RECEIVED 30/07/2024 16:50 REPORTED 30/07/2024 18:13
REF. BY Dr. ROBZ HOSPITAL Report Status Final
PATIENT HISTORY
Investigations Result Biological Ref.Int. Unit
IMMUNOLOGY
FOLIC ACID, SERUM
FOLIC ACID 3.0 Low 5.21-20.0 ng/mL
Method: (CLIA)
Comment:
Folates are a class of vitamin compounds necessary for nucleic acid and mitochondrial protein synthesist, amino acid metabolism and other cellular processes.
The predominant form of circulating folate is 5-methyltetrahydro-folic acid (5-mTHF), which requires the presence of vitamin B12 for conversion to the
metabolically active folate, ie. tetrahydrofolate (THF). Deficiency of B12 prevents cells from producing usable THF which, in turn, prevents synthesis of purines
or thymine for DNA replication and metabolization of histidine and serine. Thus, primary deficiency of folates or secondary to B12 deficiency, can lead to
megaloblastic anaemia, wherein blood cells cannot divide because they are unable to make DNA.
*** End Of Report ***
Page 3 of 4
CLIENT DETAILS Medimaa Diagnostics Pvt. Ltd.
CLIENT CODE: MED000015 A-72, Sector-50, Noida, Uttar Pradesh 201301,INDIA
MEDIMAA IMAGING PVT LTD Email: care@medimaa.in
483,RAM SHREE COMPLEX,HOSIYAR Website: www.medimaahealthcare.com
PUR,SECTOR 51 NOIDA,201301 Ph: 0120-4234948
11210022
Mobile: 8527852799 ,Ph: 8527852798
Email:
PATIENT NAME Mrs. MUKHTYARI DEVI PATIENT ID 10257199
LAB NO 11210022 AGE 75 Y GENDER Female DATE OF BIRTH
DRAWN 30/07/2024 16:48 RECEIVED 30/07/2024 16:50 REPORTED 30/07/2024 18:13
REF. BY Dr. ROBZ HOSPITAL Report Status Final
PATIENT HISTORY
Investigations Result Biological Ref.Int. Unit
IMMUNOLOGY
VITAMIN D (25-HYDROXY VITAMIN D),SERUM
VITAMIN D(25 OH) ,SERUM 48.8 Deficiency : < 10.0 ng/mL
Method: CLIA Insufficiency : 10.0-29.0
Sufficiency : 30.0 - 100.0
Toxicity : > 100.0
Comments
Vitamin D Total assay is used as an aid in the assesment of Vitamin D sufficiency in adults.
Vitamin D is acquired either by exposure to sunlight or ingestion of food containing vitamin D. It is metabolized to vit D, 25 hydroxy in the liver in the first step
by vit D,25-hydroxylase system. A small amount of it further gets metabolized by hydroxylation in kidney to vit D 1,25 dihydroxy. Since vit D, 25 hydroxy is
the predominant circulating form of Vit D in normal population, it is considered to be the most reliable index of vit D status.
Vitamin D is essential for bone health. In children, severe deficiency leads to bone-malformation, known as rickets. Milder degrees of insufficiecy are believed
to cause reduced efficiency in the utilization of dietary calcium.
The measurement of 25-OH-D is becoming increasingly important in the management of patients with various disorders of calcium metabolism associated with
Rickets, neonatal hypocalcemia, pregnancy, nutritional and renal osteodystrophy, hypoparathyroidism, and postmenopausal state.Increased levels are found in
Vit D intoxication.
Decreased levels are detected in Rickets, osteomalacia, secondary hyperparathyroidism, malabsorption of vit D (e.g. liver diseases, cholestasis), and diseases
that increase Vit D metabolism (viz. Tuberculosis, sarcoidosis, primary hyperparathyroidism).
*** End Of Report ***
Page 4 of 4
