Adobe Scan Oct 19, 2024
Adobe Scan Oct 19, 2024
Class number: _ __
N=3 me. Emroa~\t Lave ~I.!--DlliemJlau.LJ..!IUIUl~ - - -- - - - - -
Date: _ _ _ _ __
Section : \.\S~N ''\ Schedule: _ _ _ __ _ _ _ _ _ _ _ __
Materials:
Lesson title: ENVIRONMENTAL EMERGENCIES (HEAT
Electronic gadget, pen, & notebook
STROKE, FROSTBITE, ANO HYPOTHERMIA)
Learning Targets:
Al the end of the module. students will be able to·
1 Identify the environmental emergencies; Reference:
2 Describe the common causes of these environmental
emergencies: Hinkle, J. L., & Cheever, K. H. (2018).
3 Explain the clinical manifestation of heat stroke, frostbite Brunner & Suddarth's textbook of
and hypothennia; and, medical-surgical nursing (14th ed.).
4. Discuss the nursing management process of client with heal Philadelphia, PA: Lippincott Williams
stroke, frostbite and hypothermia. &Wilkins.
sr
A. LESSON PREVIEW/REVIEW
Let us have a quick review of what you have learned from the previous session. Kindly answer the posted task on the
space provided. You may use the back page of this sheet, if necessary. Here is the task:
List down at least three (3) conditions of a patient who may be at risk for developing MODS.
B. MAIN LESSON
HEAT STROKE is an acute medical emergency caused by failure of the heat-regulating mechanisms of the body.
✓- The most common cause of heat stroke is prolonged exposure to an environmental temperature of greater than
39.2C (102 SF). It usually occurs during extended heat waves, especially when they are accompanied by high
humidity.
✓- People at risk for heat stroke are those:
not acclimatized to heat
those who are elderly or very young
those unable to care for themselves
those with chronic and debilitating diseases
those taking certain medications (e.g., major tranquilizers. antichollnergics, diuretics, beta-blockers)
✓ Exertional heat stroke occurs in healthy individuals during sports or work activities (e.g., exercising in extreme
heat and humidity).
,/ Hyperthermia results because of inadequate heat loss. This type of heat stroke can also cause death.
✓ Another form of heat stroke is heat exhaustion in which the patient's temperature may be normal to 40C (104F).
The patient demonstrates weakness, hypotension, increased heart rate, and increased thirst.
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1\31
Sertion: _ __ _ _ Schedule: _ _ __
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hrn•olc:c h1 1- Clllr /
When assessing the patient, the nurse notes
the following tymptoms: .
- - profound central nervous system (CNS) ..
dysfunction (manifested by confumon , dehnum, •zarre behavior coma )
elevated body temperature (4o.s ·c [105"F] or bi '
higher)
hot, dry skin
usually anhidrosis (absence of sweating)
lachypnea , hypotension, and tachycardia
Class number: _ __
l\Jam e:
- - - - - - - - - -- - - -- - - - - - - - - - - - -
Schedule : _ _ _ _ _ _ _ _ _ _ _ _ __
Date: _ _ _ _ _ __
Section ·
Addition al measures that may be carried out vvhen appropriate include the following:
tissue to help prevent
Whirlpool bath for the affected body parts to aid circulation and debridement of necrotic
'iiiTecii ofr--
normal circulation and to
Escharotomy (incision through the eschar) to prevent further tissue damage, to allow for '
permit joint motion
- --
Fasciotomy to treat compartment syndrome
--
This document is the property ot l'hii~MA EDUtAII U (-.
·
163
i\la rne :
Class number: _ __
Section : ____ _ Schedule: _ _ _ ____ ____ ____ __
Date: _ _ _ _ _ __
After rewarming, hourly active motion of any affected digits is encouraged to promote maximal restoration
function and to prevent contractures. of
Discharge instructions also include encouraging the patient to avoid tobacco, alcohol, and caffeine
because of their
v8soconslrictive effects, which further reduce the already deficient blood supply to injured tissues
~j
1 HYPOTHERMIA is a condition in which the core (internal) temperature is 35 ' C (95' F) or less as a result
of exposure to
cold or an inability to maintain body temperature in the absence of low ambient temperatures.
✓ Urban hypothem1ia (extreme exposure to cold in an urban setting) is associated with a high mortality rate;
elderly
people, infants, people with concurrent illnesses, and the homeless are particularly susceptible.
✓ .Alcohol ingestion increases susceptibility because it causes systemic vasodilation .
✓ Some medications (e.g., phenothiazines) or medical conditions (e.g., hypothyroidism. spinal cord injury)
decrease
the ability to shiver, hampering the body's innate ability to generate body heat.
✓ Trauma victims are also at risk for hypothermia resulting from treatment with cold fluids, un-warmed
oxygen, and
exposure during examination. The patient may also have frostbite, but hypothermia takes precedence in
treatment.
Assessment and Diagnostic Findings: Hypothermia leads to physiologic changes in all organ systems.
There is
progressive deterioration with:
apathy
poor judgment
ataxia
dysarthria
drowsiness
pulmonary edema
base abnormalities
coagulopathy
eventual coma
Shivering may be suppressed at a temperature of less than 32.2C (90F), because the body's self-warmin
g
mechanisms become ineffective.
The heartbeat and blood pressure may be so weak that peripheral pulses become undetectable. Cardiac
clysrhythmias may also occur. Other physiologic abnormalities include hypoxemia and acidosis.
Managem ent consists of removal of wet clothing, continuous monitoring, rewarming . and supportive
care.
Monitoring the ABCs of basic life support is a priority.
The patient's vital signs, CVP, urine output, arterial blood gas levels, blood chemistry determinations (blood
urea
nitrogen, creatinine, glucose, electrolytes), and chest x-rays are evaluated frequently.
Body temperature is monitored with an esophageal, bladder, or rectal thermistor.
Continuous ECG monitoring is performed, because cold-induced myocardial irritability leads to conduction
disturbances, especially ventricular fibrillation .
An arterial line is inserted and maintained to record blood pressure and to facilitate blood sampling.
Rewarmin g
·- Rewarming methods include active internal (core) rewarming and passive or active external (spontaneo
us)
rewarming .
- ! i @ P tlfll8E
Class number:
--- Active internal (core) rewarming-methods are used for moderaje .to_seyere_bypotbe.rrnia_(les a '
y
[82.5"F to 90' F]) and include cardiopulmonary bypass, warm fluid administration, warm humidified oxygen
ventilator, and warmed peritoneal lavage.
- Monitoring for ventricular fibrillation as the patient's temperature i~ cre "es-fro~ 1·c to 32'C (88' F to
90' F) is
essential.
- _!:fil;si'1'.e_m acti1Le__e_~~I ~~arming is used for mild hypothermi (32.2'C lo 35· c O' F to 95'F]).
Passive active rewarming usesover-the-bed heaters to the extremities and increases blood flow to the acidotiG,
anaerobic extremities .
a
T~ cold blood ~rom peripheral tissues has high lactic acid levels. As this blood returns to the core, it causes
s1gmficant-drop m the core temperature (i.e. co,e-iemperatur-e after drop) and can potentially cause cardiac
dysrhythmias and electrolyte disturbances.
Active external rewarming uses forced air warm blankets. Care must be taken to prevent extremity burn from
these devices, because the patient may not have effective sensation to feel the burn.
1. The following clients are presented with signs and symptoms of heat-related illness. Which
of them needs to be
attended first?
manifest
A. A relatively healthy homemaker who reports that the air conditioner has been broken for days and who
fatigue, hypotension, tachypnea, and profuse sweating.
to view a parade.
8 . An elderly person who complains of dizziness and syncope after standing in the sun for several hours
n , and hot, dry ashen
C. A homeless person who is a poor historian; has altered mental status, poor muscle coordinatio
skin- and whose duration of heat exposure is unknown.
A diaphoresis, and
D. marathon runner who complains of severe leg cramps and nausea, and manifests weakness, pallor,
tachycardia.
Answer: _ __
Rationale :
... ' ' ·r; ;~ ~ ~~ ~<!'(,J·-:'i'.'\:~ ' :Pl?'Y'f "'.-~ ~-~ ts,·.
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Name .
____ _ _ __
_ _ _ _ _ Sched ule: _ _ _ _ __ ___
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2. Remov e the victim from the cold environ
3. Monitor for signs of compartment syndrome.
4 . Apply a loose, sterile, bulky dressing.
5. Administer a pain medication.
.A 5. 2. 1 3, 4
8. 2. 5. 1, 4, 3
C. 2,1, 5, 3, 4
ment.
D. 3. 2. ·1, 4, 5
,,
Answer: _ _ __
t' Rationale:
~
<
nsive and
~ the emerg ency depart ment unresp onsive , hypote
3. You are caring for a patien t who was brough t into is 106 "F. You suspe ct heat stroke . What
~ that the patien t's tempe rature
~ tachypneic. Upon furthe r asses sment, you realize
J would your priority nursin g intervention be?
j
A Obtain a history from the patient.
of treatm ent.
8 . Take oral tempe rature to monito r effecti venes s
C. Call the family for conse nt to treat.
towels and place a fan on the patient.
D. Take off the patien t's clothing, wrap him or her in wet
Answer; _ _ __
Ration ale:
_.i,j
166
-Organ
Nursing Care of Clients with life Threatening Conditions/Acutely Ill/Multi
ns, Acute & Chronic - Lecture
Problems/High Acuity and Emergency Situatio
Module #21 Student Activity Sheet
'I 1
(l,n., numbN
' •11
Date
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C Hn~l C'i'mt'!I
Ani,wttt _ _ _ _
Rationale
C LESSON WRAP-UP
IMHJI w11:, tha most useful or tile most meaningful /h,ng you havo teamed this session?
1II/Multi-Organ
Nursing Care of Clients with Life Threatening Conditions/Acutely • ct re
Problems/High Acuity and Emergency Situations, Acute & Chronic." ~h~et
Module #22 Student Activity
Class numbe r: _ __
f\l ;i fTIP
Materials:
Lesson title: STING AND BITES
Electronic gadget, pen, & notebook
Leaming Targets :
Al the end of tho module, students will be able to:
Reference:
1 Define anaphylactic shock;
2 Describe the pathophysiologic process of anaphylactic
Hinkle, J. L., & Cheever, K. H. (2018).
shock ;
Brunner & Suddarth's textbook of
3. Explain the clinical manifeGtation of anaphylactic shock;
and,
medical-surgical nursing (14th ed.).
Philadelphia, PA: Lippincott Williams
4 Discuss the nursing management process of anaphylactic
shock. &Wilkins .
A. LESSON PREVIEW/REVIEW
Kindly answer the posted task on the
Lei us have a quick review of what you have learned from the previous session.
the task:
c,pace provided . You may use the back page of this sheet, if necessary. Here is
8. MAIN LESSON
Management
is associated with sacs
Management includes stinger removal if the sting is from a bee because the venom
around the barb of the stinger itself.
The stinger is removed with one quick scrape of a fingerna il over the site.
-
~
Wound care with soap and water is sufficient for stings. _§.cmtching is avoided because
· -
it results in a histamine
169
Snake Bites
Venomnus (poisonous) snakes cause 7000 to 8000 bites in the US each year and result in 10
to 15 deaths.
Children between 1 and 9 years of age are the most likely victims. The greatest number of bites
occurs during the
daylight hours and early evening of the summer months.
The most frequent poisonous snake bite occurs from el!.YiPer.s-fCrotalidae).
The most common site is the upper extremity.
Of these bi[es-:Only 20% to25'1/o resulUne nvenomation (injection of a poisonous material by
sting, spine, bite, or
other means). .
Venomous snal<e bites are medical emergencies. Nineteen different species of venomous snakes
are found in
various regions within the US.
Nurses should be familiar with the types of snakes common to the geographic region in which
they practice.
Clinical Manifestations
Snake venom consists primarily of proteins and has a bread range of physiologic effects.
•- It may atfect multiple organ systems, especially the neurologic , cardiovascular, and respiratory
systems.
Classic clinical signs of envenomation are edema, ecchymosis and hemorrhagic bullae leading
to necrosis at the site of
envenomation
Symptoms include:
lymph node
tenderness
nausea
---!Jt111:11:li"l..,<>(t>'' ,. ·• ., . '1:.1 •,; I
Thi~ document is the p1operty 01 F'hlNMA EOUCJi.HOlll
J
Nursing Care of Cl'ient5 with Life Threatening Conditions/Acutely Ill/Multi-Organ
, Acute & Chronic · Lecture
Problems/High Acuity and Emergency Situations
Module 1122 Student Activ ity Sheet
vomiting
11umbness
;;i motallic taste in the mouth
n , hypotension, paresthesia.
h festations may progress to include fasciculatio
Wi! cut decisive treatment, these clinical mani
i;e12.ures . and coma .
---
levet..ef the
immobilizin g the i!Jju'u id body_uad..b.elow..the
ation are the priori ties of care-
--· Airway, breathing, and circul
- Ice or a tourniquet is not applie d.
-- - -
ation in the ED is performed quickly and includ
es information about the following:
in1t1al evalu ported to the ED with
nomous; if the snake is dead , ii should be trans
Whether the snake was venomous or nonve
the patient for identification.
ling the transported snake.
However. caution should be taken when hand
the snake in a stunned, not dead, and state.
Frequently , the patient and family transport
stances of the bite
Where and when the bile occurred and the circum and nearby
and symptoms (fang punctures, pain, edema, and erythema of the bite
Sequence of events, signs
tissues)
Severity of poisonous effects
Vital signs mity that was bitten
at several points: the circumference of the extre
Circumference of the bitten extremity or area
site extre mity
is compared with the circumference of the oppo
data (complete blood count , urinalysis, and coagulation studies)
Laboratory on Iha body the
s depe nd on the kind and amount of venom injected, where
Tl1e course and prognosis of snake bite injurie
and size of the patient.
bite occurred, and the general health , age,
of snake bites.
Ttiere is no one specific protocol for treatment stage .
hepa rin, and corticosteroids a(e not used during the ac:.ite
- Generally, ice, tourniquets, bite becau se they may depress antibody
6 to 8 hours after the
Corticosteroids are contraindicated in the first and used to treat
(antitoxin manufactured from the snake venom
production and hinder the action of antivenin
snake bites) . ension, their use
ension. If vasopressors are used to treat hypot
Paren teral tluids rnay be used to treat hypot ted. Typically, the patien t is observed closely
the bite is rarely indica
should be short term. Surgical exploralion of
unattended.
for at least 6 hours . The patient is never left bites.
gh envenomation 1s rare, ii can occur with snake
Administration of Antivenin (Antitoxin) althou derin g admin istration of antivenin.
toms is essential before consi
An assessment of progressive signs and symp er than 12 hours after the snake bite.
4 hours and no great
which is most effective if adrninistP.red wiihin
ATll)N
1hi, cJccurne nt is the property of PHIN MA EDUC
1ms aotornent 1s the prope 1ty of P! .!, 1/JIA c:u ULA, 1vr:
17 1
. C dltions/Acutely 111/Multl·Organ
Nursing Care of Clients with life lhreatenin:.t;;tions Acute & Chronic. Lecture
Problems/High Acuity and Emergency •Module ,#22 Student Activity Sheet
Cla~s number:
_ __ _ _ _ __ _ __ _ _ __ _ _
"'! cirn E." _ _ __ _ __
Date: ___ _ _
.::L,rtion _ _ _ _ _ Schedule · _ _ __ _ _ _ _ _ _ __ __
_
about 3% of patients
with negative skin test results develop reactions unrelafea 1oinfusio n rate.
Spider Bites
the brown recluse
There are two venomous spiders found in the United States that typically interact with humans:
-
and the black widow.
places such as closets, woodpiles . and attics, as well as in shoes.~
Both are usually found in dark
~ sinless.
within 24 to 72 hours.
- Systemic effectSSLi"cfi"as fever & chills. nausea & vomiting malaise, and joint pain develops
Ne~rosis oc~urs in the next 2 to
The site of the bite may appear ~ i t h 1 n~ r the bite.
4 days m approximately 10% of cases. Ih,e center-of tbe6ite-may become
necrotic, and surgical debndement may be
treatments may be helpful.
necessary. Wound care consists of cleansing with soap and water, and hyperbanc oxygen
\og \,.V' p..1.,v• \ t'\""
Class number: _ __
I\Jame·
Date : _ _ _ _ __
Sectirm · - - - - - Schedule : - - - - -- - - - - - - - - - -
Most wounds heal within 2 to 3 months. Black widow spider bites feel like pinpricks. Systemic
effects usually occur
within 30 minutes-much more rapidly than with brown recluse spider bites.
Treatn:i~nt in~olv~s _applicaUon of ice to the site to decrease systemic toxin delivery. Cardiopulmonary
monitoring is
essent1a1. Antivenin Is effective for black widow spider bites. This antivenin is horse serum-ba
sed; therefore, testing for
sensitivity must be performed prior to administration.
Tick Bites
This is common in many areas of the US, and they usually occur in grassy or wooded areas.
It is important to learn the place where the bite occurred as well as the location of the bite on
the body.
The patient may demonstrate weakness ; joint pain; skin rash, especially on the palms and soles
of feet;
headache; and fever.
Ticks can carry diseases such as Rocky Mountain spotted fever, tularemia, and Lyme disease.
The tick bite itself is not usually the problem; rather, it is the pathogen transmitted by the tick
that can cause
serious disease.
The tick should be removed , and the patient should be informed of the signs and symptoms
of diseases carried
by ticks, especially if the patient lives in an area endemic for tick-related diseases (e.g., Lyme
disease).
Lyme disease has three stages:
Stage I presents with a "bull's eye" rash (i.e., erythema migrans) that typically can be found in
the axilla, groin, or
thigh area and that appears within 4 weeks after the tick bite, with a peak manifestation time
of 7 days after the
bite. Classically, this rash is at least 5 cm in diameter with bright red borders.
0 It is accompanied by flulike signs and symptoms that may include chills, fever, myalgia, fatigue,
and
headache. Without treatment , the rash subsides within 3 to 4 weeks.
o However, the rash and flullke manifestations can be signlficanUy reduced within days if prompt
treatment
with antibiotics is initiated. If antibiotics are not administered,
Stage II Lyme disease may present within 4 to 10 weeks following the tick bile and may manifest
with joint pain,
memory loss, poor motor coordination, and meningitis . . .
Stage Ill can begin anywhere from weeks to more than cl year after the bite and has senous .
long-term chronic
sequelae, including arthritis, neuropathy, myalgia, and myocarditis.
~ /'
~
F~
I 1"
Name.
Class number: _ __
6 A camp nurse is providing snakebite prevention tips. Which statement indicates a need for additional education?
a. "'A dead snake is a safe snake."
b. "Snakes are most active on warm nights."
c. "Snakes should be transported in sealed glass containers."
<i "Venomous snakes are not good pets."
Answer: _ _ __
Rationale:
7. A 12-year-old client comes to the emergency department (ED) after being bitten by a scorpion at a local petting zoo.
Which action does the nurse perform first?
A. Administers tetanus shot
8. Applies an ice pack to the sting site
C. Assesses the client's vital signs
D. Calls the poison control center
Answer: _ _ __
Rationale:
8 A nurse is teaching a class of park rangers-in-training about prioritizing care for clients who have received snakebites.
Which ranger's statement demonstrates a need for further clarification'?
A. "Do not allow the client to ingest any alcohol or caffeine."
B. "The extremity should be kept below the level of the heart."
C. "'The first priority is to move the client to a safe area away from the snake."
D. "You should ti rst place a tourniquet above the bite."
Answer: _ _ __
Rationale:
i 7$
9 While on the playground, a school child is stung by a bee, resulting in redness and swelling. The school nurse is nearby
,,hen It happens. What does the nurse do firnt?
A .l\pp!ies an ice pack to the stinger
B. Gently scrapes out the stinger with a credit card
C Injects the child with an epinephrine pen (Epi-Pen auto-injector)
D. Removes the bee and saves it for evidence of the sting
Answer: _ __
Rationale:
10. A young man is brought lo the Emergency Department after receiving multiple fire ant bites while working in his yard.
Although initially alert and oriented, he begins to develop wheezing and on itchy throat. He complains of nausea and
severe anxiety. The ED nurse should prepare to administer all of the following for initial treatment , except:
A Adrenaline C. Oxygen
B. An tibiotic D. Antihistamines
Answer: _ __
Rationale:
C. LESSON WRAP-UP
CAT 3-2-1
This strntegy provides a structure for you lo record your own comprehension and summarize your learning. Let us see
your progress in this chapter!
1.
2.
2.
1.
Class number: _ __
N;ime .
- - -- - -
Date: _ _ _ _ _ __
:,Prt ,on· - -- - - Schedule:
Materials:
Lesson title: POISONING, NEAR DROWNING AND
Electronic gadget. pen. & notebook
DECOMPRESSION SICKNESS
Learning Targets:
Reference:
.LI.I the end of the module, students will be able to·
1. Describe the effects of poison lo a patient's body;
Hinkle. J. L., & Cheever. K. H. (2018).
2. Enumerate the immediate emergency treatment of poison,
Brunner & Suddarth 's textbook of
3 Define near drowning;
4 Discuss the therapeutic management goals of drowning:
medical-surgical nursing (14th ed.).
Philadelphia, PA: Lippincott Williams
and.
&Wilkins.
"''' b. Explain assessment findings of decompression sickness.
A. LESSON PREVIEW/REVIEW
task on the
Let us have a quick review of what you have learned from the previous session. Kindly answer the posted
5:pc1ce provided You may use the back page of this sheet. if necessary. Here is the task:
ij
8. MAIN LESSON
POISONING
the body in
✓ A poison is any substance that, when ingested, inhaled, absorbed, applied to the skin, or produced within
relatively small amounts, injures the body by its chemical action.
a major
✓ PoisQDingJro1DlD.haJgtion and inges~on of toxic materials.J:1ot~ intentional and unintentional, constitutes
,/ health hazar~ aQ_,emergency, sj t1,1atioJ1. -
111/MLllti-Organ
Nursing Ca " 0 f Cl' Condl tions/AcutelY nlc • Lecture
r• rents with Life Threatening
Situations, Acute & ct,roctlllit'Y Sheet
Problems/liigh Acuity and Emergency
Module #23 Stude nt A
Name :
- -- - -- Class number: - - -
SPctron: Schedule:
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its absorption.
Measures are instituted to remove the toxin or decrease
can be a strong acid or alkaline substance, is given water
- The patient who has ingested a corrosive poison , which
or milk to drink for dilution.
airway edema or obstruction or if there is clinical
- However. dilution is not attempted if the patient has acute tion.
evidence of esophageal. gastric, or intestinal burn or perfora
as prescribed:
Th€! following gastric emptying procedures may be used
Syrup of ipecac to induce vomiting 111 the alert patien t (o.ever use-with-corrosLve poisons)
saved and sent to t~ ratory for testing (toxicology
Gastric lavage for the obtunded patient; gastric aspirate i5
screens)
is absorbed by charcoal
Activated charcoal administratio n if the poison is one that
Cathartic, when appropriate
te), it is administered as early as possible to
If there is a specific chemical or physiologic antagonist (antido
reverse or diminish the effects of the toxin.
If this measure is ineffective. procedures may be
initiated to remove the ingested substance.
s),
of charcoal, diuresis (for substances excreted by the kidney
These proce dures 111clude administration of multiple doses
dialysis, er hemoperfusion.
sing it through an extracorporeal circuit and an
Hemoperfusion involves detoxification of the blood by proces
the cleansed blood is returned to the patient.
adsorbent cartridge containing charcoal or resin. after which
and fluid and electrolyte balance are monitored closely .
Throughout detoxification, the patient's vital signs, CVP,
Hypotension and cardiac dysrhithmias are possible.
the poison or from oxygen deprivation. If the patient
Seizures are also possible because or CNS stimulation from
complains of pain, analgesics are administered cautiously.
on or normal physiologic functions.
Severe pain causes vasomotor collapse and reflex lnhibiU
is imminent, written material should be given to the
After the patient's condition has stabilized.and discharge
ms related to the poison ingested and signs or
patient indicating the signs and symploms of potential proble
sympioms requiring evaluatiori by a physician
attempt, a psychiatric consultation should be requested
If poisoning was determined to be a suicide or self-ha rm
before the patient is discharged.
and home poison-proofing instructions should be
In cases of inadvertent poison ingestion, poison prevention
provided to the patient and family.
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17 (l
Date: _ __ _ __ _
~-,-rtio r, - - -- -- Schedule :
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Clinical Manifestations:
Because the CNS has a critical need for oxygen, ~ ptoms predomiAate.withJ;arbon mowxida.toxicitt_
A person with carbon monoxide poisoning may ae pe~r intoxicate d (from cerebral hypoxia).
Other signs and symptoms include headache , muscu ar weakness . palpitation . dizziness , and confusion , which
can progress rapidly to corna.
sign.
Skin color, which can range from pink or cherry-red to cyanotic and pale, is not a reliable
because the hemoglob in is well saturated . It Is not saturated with oxygen, but the
Pulse oximetry is also not valid.
carbon monoxide
pulse oximeter indicates only if the hemoglobin is saturated. in this case. ii is saturated with
rather than with oxygen.
Managem ent
Exposure lo carbon monoxide requires immediate treatment.
. Goals of management are to reverse cerebral and myocardial hypcm_a _amLto hasten eliminatio
- - -
n of car:boo._
mo.noxide.
Whenever a patient inhales a poison, the following general measures apply:
- Carry t~e patient to fresh air immediately; open all doors and windows
L0osen all tight clothing.
Initiate cardiopulmonary resuscitation if required: administer 100% oxygen.
Prevent chilling; wrap the patient in blankets.
Keep the patient as quiet as possible.
with carbon
Do not give alcohol in any form or permit the patient to smoke. In addition, for the patient
and before treatment with
monoxide poisoning, carboxyhemoglobin levels are analyzed on arrival at the ED
oxygen if possible.
hypoxia and
100% oxygen is administered at atmospheric or preferably hyperbaric pressures to reverse
accelerate lhe elimination of carbon monoxide.
. Oxygen ii; administered until the carboxyhemoglobin level is less than 5%. The patient is
visual disturban ces, and deterioration of mental
monitored continuously .
status and behavior may
Psychoses, spastic paralysis, ataxia,
persist after resuscitation and may be symptom s of permanen t brain damage.
. When unintentional carbon monoxide poisoning occurs. the health department should be
contacted so that the
if poisoning was
dwelling or building in question can be' inspected. A psychiatric consultation is warranted
r
determined to be a suicide attempt.
Class number: _ __
_ _ __ Schedule: Date: _ _ __ __
------------- ---
✓ The skin ol health care personnel assisting the patient should be ~ppropnately protected if the burn 1s extensive or if
the agent is significantly toxic or Is still present.
" Prolonged lavage with generous amounts of tepid water is important. Attempts to determine the identity and
characteristics of the chemical agent are necessary in order to specify future treatment. .
' The standard burn treatment appropriate for the size and location of the wound (antimicrobial treatment, debndement,
tetanus prophylaxis, antidote administration as prescribed) is instituted.
✓ The patient may require plastic surgery for further wound management .
,/ The patient is instructed to have the affected area reexamined at 24 and 72 hours and in 7 days because of the nsk of
underestimating the extent and depth of these types of injuries.
FOOD POISONING
" This 1s a sudden illness that occurs after ingestiol'l of contaminated food or dnnk.
< Botulism is a serious form of food poisonin3 that requires continual surveillance.
✓ The key to tn'!atment is determining the source and type of food poisoning .
./ If possible the suspected food should be brought to the medical facility and a history obtained from the patient or
family
Food, gastric contents. vomitus. serum. and feces are collected for. examination.
The patient's respirations, blood pressure, level of consciousness. central venous pressure (CVP) lif indicated).
and muscular activity are rnorntored closely.
Measures are instituted to support the respiratory system. Death from respiratory paralysis can occur with
botulism. fish poisoning, and some other food poisonings.
Because large volumes of electrolytes and water are lost by vomiting and diarrhea, fluid and electrolyte status
should be assessed.
~rniling.p@uces alkalosis, and seyem-diarrhea-pr~doces-aeidosis-:-
Hypovolemic shock may also occur from severe fluid and electrolyte losses.
M~nag emenl:
An antiernetic medication i5 administnred parenlerally as prescribed if the patient cannot tolerate fluids er
rnP,d ications by mouth.
For mild nausea, the patient is encouraged to take sips of weak tea, carbonated drinks, or tap water.
After nausea and vomiting subside, clear liquids are usually prescribed for 12 to 24 hours, and the diet is
gradually progressed to a low-residue, bland diet.
NEAR DROWNING
./ This is defined as survival for at least 24 hours after submersion that caused a respiratory arrest.
✓ The-most com~ BAGEHfr·hypoxei+lia.
✓ Drowning is the second most commQJ1 cause of unintentional death in children younger than 14 years.
o An estimated 8000arcwnings and 90,000 near drownings occur yearly in the United States.
Suicide by drowning rarely occurs in pools and rarely involves alcohol. Efforts lo save the patient should not be
abandoned premature!}'. Successful resuscitation with full neurologic recovery has occurred in near-drowning patients
after prolonged submersiol'\ in cold water. This is possible due to decrease in metabolic demands and/or the diving reflex.
If there is a violent struggle associated with the near-drowning episode, exercise-induced acidosis and tachypnea can
result in aspiration. Hypoxia and acidosis cause eventual apnea and loss of consciousness. When the victim loses
consciousness and makes a final effort to breathe, the terml_r.@..9.aSp_gccurs. Water then moves passively into the
airways prior to death. After resuscitation, hypoxia a nd acidosis are the Qrimary complications experienced by a person
J!'.!io h_g.5 oea~wned; immediate intervention irrihe ED is essential.
Resultant pathophysiologic changes & pulmonary injury depend on type of nuid (fresh or salt water) & volume aspirated.
- ,Fresh water aspiration results in a loss of surfactant and, therefore, an inability to expand the lungs.
~ ,,.,.f-,,_ .ft;•!:- S ~ 1:.espiration leads to ~ _ f r o m the osmotic e~cts oJ the salt within the I~.
~ - IH!l"'person survives submersion, acute respiratory distress syndrome (ARDS). resulting in hypoxia. hypercarbia,
and respiratory or metabolic acidosis, can occur.
Management: Therapeutic goals include maintaining ~.hraLpe~ on and adequate oxygenation to prevent further
damage to vital organs.
,j Immediate cardiopulmonary resuscitation is the factor with the greatest influence on survival.
- The most important priority in resuscitation is to manage the hypoxia, acidosis, and hypothermia.
'
j
Prevention and management of hypoxia are accomplished by ensuring an adequate airway and respiration, thus
improving ventilation (which helps correct respiratory acidosis) and oxygenation.
Arterial blood gases are monitored to evaluate oxygen, carbon dioxide, bicarbonate levels, and pH. These
parameters determine the type of ventilatory support needed.
Use of endotracheal intubation with PEEP improves oxygenation, prevents aspiration, and corrects
intrapulmonary shunting and ventilation-perfusion abnormalities (caused by aspiration of water).
If the patient is breathing spontaneously, supplemental oxygen may be administered by mask However, an
endotracheal tube is necessary if the patient does not breathe spontaneou~ . Because of submersion, the patient
is usually hypothermic. '
A rectal probe is used to determine the degree of hypothermia.
Class number: - -
:rr:rtn n ·
---- Schedule: Date: _ _ _ __ _
--- - - --- ------ -
Prescribed rewarming procedures {e.g . extracorporeal warming,
warmed peritoneal dialysis, inh~lation of warm
aorosohzed oxygon, torso warming) are started during resuscitation.
The choice of wanning method is determined by the severity and
duration of hypothermia and available
resources.
lntravascular volume expansion and inotropic agents are used to
treat hypotension and impaired tissue perfusion.
ECG monitoring is initiated . because dysrhythmias frequently occur.
An indwelling urinary catheter is inserted to measure urine output.
Hypothermia and accompanying metabolic acidosis may compro
mise renal function .
Nasogastric intubation is used to decompress the stomach and to
prevent the patient from aspirating gastric
contents.
DECOMPRESSION SICKNESS
✓ This is also called ~ s Rds'.', occurs in patients who have engage
d in diving (lake, as well as ocean, diving), high-
altitude flying , or flying in commercial aircraft within 24 hours after
diving.
✓ It occurs relatively infrequently in the United States, but its effects
can be hazardous. Being aware of decompression
sickness and assessing the patient properly ensures proper manag
ement and results in the least morbidity possible.
✓ Decompression sickness results from !prmation of nitr~en bu,bble
s that occur with rapid changes in atmospbe~
E,l'essure.
✓ They may occur in joint or muscle spaces. resulting in musculoskele
tal pain, numbness, or hypesthesia.
✓ More significantly, nitrogen bubbles can become air emboli In lire.blo
odstream and thereby produce stroke, paralysis,
or death.
-
✓ Taking a rapid history about the events preceding the symptoms
is essential.
•' Recompression is necessary as soon as possible and may necess
itate a low-altitude flight to the nearest hyperbaric
chamber.
I
I
l
I
182 I
Class number: _ __
~cctron:
- -- - - Schedu le: Date.
- -- - - - -- - - -- - - - - -
Ma"lagement
A pa.t~nt airway and adequate ventilation are established, as described previously, and 100% oxygen is
admrnrstered throughout treatment and transport.
A ~hest x-ray is obtained lo identify aspiration, and at least one IV line is started with lactated Ringer's or normal
saline solution.
The cardiopulmonary and neurologic systems are supported as needed. If an air embolus is suspected, the head
of the becLshould--be•lowered..
The patient's wet clothing is removed, and the patient is kept warm.
Transfer to the closest hyperburic chamber for treatment is initiated. If air transport is necessary, low-altitude night
(below 1000 feet) is required.
However, the patient who is awake and alert without central neurologic deficits may be able to travel by ground
ambulance or by automobile, depending on the severity of symptoms.
Throughout treatment, the patient is continually assessed, and changes are documented. If aspiration is
suspected, antibiotics and other treatment may be prescribed.
1 Nurse Kelly is teaching the parents of a young child how tc, handle poisoning. If the child ingests poison. what should
!he parents do first?
A. Call an ambulance Immed1ately C. Punish the child for being bad.
8. Call the poison control center. D. Administer ipecac syrup.
Answer: _ _ __
Rationale:
2. A client arrives in the emergency unit and reports that a concentrated household cleaner was splashed in both eyes.
'v'l/111ch of the following nursing actions is a priority?
A Use Restasr:; (Allergan) drops ,n the eye.
B Flusl1 the eye repeatedly using sterile normal saline
C Examine the client's visual acuity.
D. Patch the eye.
Answer: _ _ __
Rationale.
Class number: _ __
Name
D.ite: _ _ _ _ _ __
Section· _ _ __ __ Schedule: _ _ _ _____ _____ __ _
-1 fhe nurse employed in an emergency department is assigned to triage clients coming to the e~ergency department for
treatment on the evening shift. The nurse should assign priority to which client?
A A client complaining of muscle aches, a headache, and history of seizures
B A client who twisted her ankle when rollerblading and is requesting medication for pain
C. A client with a minor laceration on the index finger sustained while cutting an eggplant
D. A client with chest pain who states that he iust ate pizza that was made with a very spicy sauce
Answer: _ _ _ _
Rationale :
5 Which of these toxic substances is more likely to cause sickness in infants and elderly adults?
A. Lead D Bleach
B Carbon monoxide E Turpentine
C Bee venom
Answer: _ _ __
Rationale:
6. How can you reduce the risk that a child will be accidentally poisoned by medicine?
A. Never say that medicine is "candy"
B Keep medicines in their original containers and in locked cabinets
C. Ma1'c ~ure you put medicines away after using them
' D. Ail of the above
Answer: _ __
Rationale:
7. How can you reduce your risk for accidental poisoning with medicine?
1 A Always turn on t11e light when tak111g medicine
B. Clean out your medicine cabinet regularly
C Carefully read the labels on your medicine
0 . All of the above
Answer: _ _ __
Rationale:
8 If your child eats or drinks a toxic substance. what should you do?
A Call tile poison control center right away C Call your child's healthcare provider
B Tri to gel }'"OUr child to throw up (vomit) D None of the above
Answer: _ _ _ _
Rationale:
.., ~-- - ~ ~ :i.:
~~
;
~ ' t
1[ld
l-Organ
atening Conditions/Acutely 111/Mult
Nursing Care of Clien ts with Life Thre ure
cy Situations, Acute & Chronic - Lect
Problems/High Acu ity and Emergen She et
Module #23 Student Activity
Date: _ _ _ _ _ __
Sec t ion. Scl1edul e
rol center?
e ready when you call a poison cont
9 V\'.hal information should you hav
A T1111e the po1s0111ng occurred
B Age of your child
C Name of the product taken
D. All of the above
Answer: _ __ _
Rationale:
ucts ?
dental poison,n g by household prod
10 How can you help prevent acci
A Never mix household chemic3I
products together
a fan when using a chemical product
S. Open a window or turn on
what's inside
C Nev er sniff containers lo find out
0 All of the above
Ans wer: _ _ __
Rationale:
C. LESSON WRAP-UP