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Intestinal Obstruction

The document provides a comprehensive overview of bowel obstruction, including definitions, causes, pathophysiology, presentation, and management strategies. It distinguishes between small and large bowel obstructions, detailing symptoms, diagnostic methods, and treatment options, including both conservative and surgical interventions. Additionally, it discusses complications associated with bowel obstruction and outlines the nature of presentations, emphasizing the importance of timely diagnosis and management.
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0% found this document useful (0 votes)
3 views7 pages

Intestinal Obstruction

The document provides a comprehensive overview of bowel obstruction, including definitions, causes, pathophysiology, presentation, and management strategies. It distinguishes between small and large bowel obstructions, detailing symptoms, diagnostic methods, and treatment options, including both conservative and surgical interventions. Additionally, it discusses complications associated with bowel obstruction and outlines the nature of presentations, emphasizing the importance of timely diagnosis and management.
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as PDF, TXT or read online on Scribd
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GENERAL

SURGERY
Dr. Laith Nawafleh
Definition o High small bowel obstruction → greenish-blue, bile-stained
o Complication: electrolytes imbalance, hypovolaemic shock,
 Partial or complete Blockage in large or small intestine that results in Respiratory complications
the failure of intestinal contracts to pass food or liquid.  Constipation
o More distal obstruction  earlier constipation
Etiology (causes) o classification
 relative Constipation: no feces (only flatus is passed)
 small intestinal-obstruction  absolute Constipation: (Obstipation): no flatus, no feces
 in adult ( adhesion (most common ) / strangulated hernia /CRC)  Distention
 in children (strangulated hernia ( most common )) o More distal obstruction  earlier distention
 infants (Intussusceptions (most common )) o Visible peristalsis may be present
 in neonates (congenital (atresia, volvulus neonatorum, imperforate
anus )) Physical examination
 large intestinal obstruction  most common cause : CRC
 General
Pathophysiology  hypotension
 Signs of dehydration and electrolyte loss (dry
 The bowel proximal to the obstruction: initially  peristalsis → dilation skin and tongue, poor venous filling, sunken
by fluid, gas, secretions →  peristalsis → flaccidity and paralysis eyes and oliguria)
 The bowel distal to the obstruction: normal peristalsis and absorption  Inspection
until it become empty → collapse  Distention, hernias, scars, visible peristalsis
 NOTE: (PYQ) (pic)
 Collapsed caecum → small intestinal obstruction  Palpation
 Distended caecum → large intestinal obstruction.  guarding, rigidity
 tenderness, mass → (tumors or
Presentation (HISTORY) intussusception)
 Auscultation
 4 Cardinal symptoms  frequent and high pitched (early) VS absent
 Colicky abdominal pain. (late/ paralytic ileus)
o first (earliest) symptom  DRE  obstructive mass in the pouch of Douglas
o More severe and continues pain likely of strangulation.
o It may relieve by vomiting. Investigation
o NOTES
 Simple occlusion  colicky intermittent pain  Labs
 Strangulation  sever pain  urea, creatinine and electrolyte (for dehydration), CBC (anemia)
 Paralytic ileus  no pain  Imaging
 Vomiting  x-ray
o More proximal obstruction  earlier vomiting o Erect x-ray  detect air fluid level
o Pyloric obstruction → watery and acid.  small intestine obstruction  stepladder pattern

1
o Supine x-ray (more accurate) distended loops that indicate  CT
obstruction o Localize the site of obstruction, detect
 High obstruction (small intestine)  centrally position, lesions and colonic tumors, and may
Vulvulae coniventae diagnose unusual hernias.
 Low obstruction (large intestine)  peripherally position,
Haustrations which do not extend across the whole width
o 369 rule (PYQ) SUMMERY
 Small bowel: <3 cm
 Large bowel: <6 cm High SBO Low SBO LBO
 Cecum: <9 cm Periumbilical pain (above) Periumbilical pain (bellow) Peripheral pain
Early vomiting & Late In between Early constipation &
constipation Late vomiting
Minimal central distention Moderate Central Early peripheral
distention distention
x-ray finding x-ray finding x-ray finding
1. no air-fluid level 1. multiple air-fluid level 1. fewer air-fluid level
2. centrally position 2. centrally position 2. peripherally position
3. Vulvulae coniventae 3. Vulvulae coniventae 3. Haustrations
NOTE:
Gastric outlet obstruction (PYQ)
- vomiting of undigested food, weight loss sometimes
- physical findings : Succession splash, Visible peristalsis, wasting
& dehydration
- causes : PUD, Gastric CA, Pancreatic pseudo-cyst

Management
 X-RAY- “Barium Follow-Through”  Conservative
o Patient drinks a contrast medium containing  Indication
barium sulfate o Distinction from postoperative paralytic ileus is uncertain.
o Contrast medium  white on x-rays o Obstruction resulted from massive intra abdominal adhesions
o The test is completed when the Barium is rendering Surgery dangerous
visualized at the Caecum o Chronic obstruction
 Barium Enema  Initial management (PYQ)
o Injecting a barium sulfate dye through the o NPO
back passage (rectum) o Gastrointestinal decompression via a nasogastric tube (NGT)
o Taking x-ray views of bowel as the dye fills o IV fluid
the large bowel. o Fluid and electrolyte replacement
o Antibiotics: if Strangulation is found or suspected , and for all
patients undergoing surgery

2
 SURGERY (Laparotomy)
 Absolute indication
Classification (according to 
o Peritonitis Location
o Visceral perforation
o Irreducible hernia  Small intestine
o intestinal strangulation  high small intestinal: duodenum or jejunum
 Relative indication  low small intestinal: ileum
o Palpable mass lesion  Large intestine
o 'Virgin' abdomen
o Failure to improve Etiology
 Bowel is inspected and non-viable bowel is removed (PPP-CC)
 Mechanical (dynamic)  intraluminal, intramural, extramural
 Intraluminal
o Fecal impaction
o foreign body
o gallstone ileus (very-rare)
 tends to occur in elderly
 large gallstone directly go through the
gall bladder into the duodenum
o if viable : reduce to the abdomen  most common site (Ileocecal valve)
o if Non-viable : (the surgical procedure according to site):  triad of
 small intestine or right colon :→ resection with  small bowel obstruction
anastomosis.(because less number of bacteria found here)  a gallstone outside the gallbladder ( mainly in the RIF)
 left colon : Hartmann’s technique  air in the bile ducts (pneumobilia)

 Specific Management ‫كل هاي المواضيع رح نحكي عنها تحت‬


o bezoars
 Hernia → Operation (herniotomy , hernioraphy , hernioplasty)
 firm masses of undigested hair ball and
 Adhesions → Conservative first then adhenolysis
fruit/vegetable fibers
 Volvulus → Untwisting and or operate
 associated with an underlying psychiatric
 Mesenteric ischemia → Anti-coagulant , Embolectomy ,
abnormality
Revascularization , Colectomy
 Abscess or Peritonitis → Drain and Treat
 Intussusception → Pneumatic or Barium
Reduction or Operate o Worms
 Strangulation in large bowel (after splenic  Ascaris lumbricoides
flexure)
o Hartmann’s procedure

3
 Intramural EXAMPLES
o Stricture (mostly due to TB or IBD)
 adults  sigmoid volvulus (PYQ)
o Malignancy (apple core sign on x-ray)  Barium enema
o intussusception  x-ray: Omega loop (grossly
 occurs when one portion of the gut invaginates dilated sigmoid colon / coffee
into an immediately adjacent segment bean sign)
(telescoping).  definitive tt : surgery (operative
 Almost always, it is the proximal into the distal. reduction)
 90 % is idiopathic  complication : bowel ischemia and perforation
 more common in children
 pediatrics  midgut volvulus (PYQ)
 Site
 Malrotation of the midgut
 Children: mostly ileocolic
 Adult : mostly colocolic  life-threatening
 Causes  history of 6 week old child Vomits (bile-
 Children: Meckel’s diverticulum, polyp stained blood)
 Adult: polyp (Peutz–Jeghers syndrome), submucosal lipoma  diagnosis
or other tumors  CT: whirloop sign
 Diagnosis  Barium swallow (gold standard): corkscrew sign
 clinical presentation (PYQ)
severe paroxysmal colicky abdominal
pain with currant jelly stool (mixture of
mucus and blood).
Bilious vomiting in infants, abdominal
distention, fever + shock, sausage
shaped mass palpable in abdomen
 Investigation :  Extramural
Barium: Claw’ sign of o Adhesion
iliac intussusception  40% of all common causes of
US: target sign obstruction
 75% of small bowel obstruction cases
o Volvulus (MCC)
 twisting or axial rotation of a portion of bowel  Most commonly caused by previous
about its mesentery (rotation) surgery
 obstruction to the lumen (>180° torsion)  Prevention
 obstruction and strangulation (>360 torsion)  Good surgical technique
 Types:  washing the peritoneal
- Primary due to congenital malrotation of the gut or cavity with saline to
congenital bands. remove clots
- Secondary form (more common) secondary to rotation  Minimizing contact with gauze
around an acquired adhesion or stoma  Treatment: adhenolysis
4
o Band  Pseudo-obstruction
 Types: o Autonomic imbalance resulting from
-Congenital: (obliterated vitellointestinal duct) decreased parasympathetic tone or
-Acquired: String band following previous excessive sympathetic output
bacterial peritonitis. o Usually in the colon
-portion of greater omentum o Ogilvie's syndrome (Acute colonic
o Hernia pseudo-obstruction)
 External hernia, through abdominal wall  Affects large intestine
 Internal hernia, rare  Dilated colon
o Neoplasm  Tx: conservative, Erythromycin, IV
neostigmine, surgery
 Functional: (adynamic)
 Paralytic Ileus
 Hirschsprung disease o failure of transmission of peristaltic waves
o congenital megacolon characterized by the absence of secondary to neuromuscular failure
myenteric and submucosal ganglion cells in the distal o causes (5Ps+drugs)
alimentary tract  Postoperative duration of 24–72 hours.
o 1 per 5000 live births.  Peritonitis
o 4 times more common in males than females.  Pelvic/spinal fractures (reflex ileus)
o Types ‫قراءة‬  low Potassium (hypokalaemia ).
 Classical HD (75% of cases): Rectosegmoid area and distally  Pseudo-obstruction
 Long segment HD (20% of cases): any part of the colon  Drugs: Tricyclic antidepressants,
beyond the recto sigmoid area is affected. Lithium therapy, Excessive opiate use
 Total colonic aganglionosis (3-12% of cases): Affects the and Anti-cholinergics
entire colon and often extends to the terminal ileum.
 Mesenteric artery occlusion
 Rare variants include the following:
 Total intestinal aganglionosis: it’s incompatible with life
because the whole GI tract don’t have ganglion
 Ultra-short-segment HD: limited to internal anal Degree of obstruction
sphincter
 Complete: No passage of luminal contents beyond the
o Management:
surgery obstruction point
 Partial: Some intestinal contents pass through

 Note:
closed loop obstruction: the bowel is obstructed at
both the proximal and distal points (seen in volvulus)

5
Complications
 Simple
 Intact blood supply
 Hyperperistalsis to overcome the obstruction → colics
 Powerful antiperistalsis → vomiting
 Complicated
 Compromised blood supply (Strangulation)
 Features of strangulation (PYQ)
• General: tachycardia, pyrexia, toxic appearance, leukocytosis
• Pain becomes constant rather than colicky.
• Signs of peritonitis: abdominal wall rigidity, absent bowel sounds,
and can lead to toxic or septic shock

 Paralytic ileus: Paralysis of intestinal movements  Neurogenic


obstruction THE MOST common form

Nature of presentation
 Acute: Sudden onset, seen usually in SBO
 Chronic: Gradual onset, seen usually in LBO
 Subacute: On and Off obstruction

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