304 Ippi
304 Ippi
DISSOC. IDENTITY DO - 2 or more personality 1%,underdiagnosed - trauma, e.g. - hippocampus - coping - memory - chronic and -2 or more distinct -EEG – r/o seizure
state or identities abuse or (memory) mechanism to lapses disabling identities, recurrent DO cause memory
-accompanied by memory -MC w/ history of neglect compartmentalize - changes in - OT memory gaps, gaps
gaps trauma disrupting - amygdala trauma affect goal:integrate significant distress,
identity (emotional - black outs identities not attributable to Med: SSRI –
integration processing) - inconsistent substances or other fluoxetine
w/ usual medical conditions To manage cc
personality depression or
anxiety, does not
treat DID directly
DISSOC. AMNESIA -inability to recall - 2-6% -psych - hippocampus - defense - memory gaps - resolved when -inability to recall -MRI – r/o brain
autobiographical info. -associated w/ response to (memory) mechanism to avoid - confusion e person is autobiographical structures causing
Typically related to trauma traumatic experiences trauma leading stress by - occasionally removed from e info. Related to memory issues
to selective - amygdala repressing wandering trauma trauma, significant
memory (emotional traumatic memories (fugue) distress or fx -benzodiazepines
blocking processing) impairment, not d/t e.g. lorazepam –
substance or other assists in recalling
medical conditions. memories
DEPERSONALIZATION -detachment from -1-2% - linked to -sensory - defense to reduce -unreality -episodic or -persistent or -EEG – r/o seizure
DEREALIZATION oneself / environment with - associated w/ high trauma and perception emotional distress -detachment chronic current experiences DO or temporal
intact reality testing stress situations or stress areas of e brain by feelings of from oneself -can be lifelong of lobe epilepsy
trauma - triggered by -prefrontal detachment -dream-like depersonalization,
substance cortex (self- surroundings derealization, or -SSRI – sertraline
abuse awareness) both, w/ distress or for assoc.
fx impairment depression and
anxiety
DISSOC. FUGUE -unexpected travel away -rare -triggered by Memory - defense -wandering -short-lived, may - specifiers involving -Toxicology –
-inability to recall one’s -associated w/ acute stress or processing mechanism by -confusion recover memory travel and identity screening to r/o
past extreme stress trauma areas of e brain escaping from -inability to eventually confusion drug-induced
- identity confusion distressing recall personal states
situations through details
amnesia -assumes new -no treatment,
identity psychotherapy is
preferred
- incongruence between W= .002% -.003% -genetic - brain structure -brain structure -desire to live -improved - 6 months -Hormonal Level
one’s M= .004 %- .014% -hormonal -hormonal -hormonal as another outcomes in Assessment - for
experienced/expressed - psychosocial influences influences gender supportive those seeking
gender and assigned factors -discomfort w/ environment hormonal therapy
gender w/significant one’s own body -sx may persist
GENDER DYSPHORIA distress -social distress or resolve -hormones e.g.
testosterone and
estrogen; risk =
mood changes
and cardiovascular
issues
-distressing somatic sx w/ -4-6 % -cognitive -amplification of -resistant to -often chronic -persistent somatic Basic Diagnostic
excessive thoughts or distortions sensations reassurance but treatable in sx for over 6 months Tests e.g. CBC –
behaviors focused on sx -heightened -maladaptive -physical therapy w/ excessive to r/o med
SOMATIC SYMPTOMS DO bodily thought patterns complaints thought/beh related conditions
awareness involving to these sx SSRI – fluoxetine
-psychological multiple body For anxiety/ depr.
factors systems
DEFINITION EPIDEMIOLOGY ETIOLOGY BMS PATHOPHYSIO S/SX C&P DSM LAB & MED
BIPOLAR I Characterized by manic- - trauma, e.g. Neurotransmitters NE – decreased, -P prognosis Medications:
depressive episodes abuse or correlated with than MDD -Depressive
neglect depression -40-50% may Disorders or
disrupting have 2nd manic depressive
identity Serotonin – low episode within episodes
integration levels in CSF and the 2 years after
reuptake sites on the first episode = SSRI (first line)
platelet -high changes of
suicide =NE Reuptake
Dopamine – -chronic, social Inhibitors
reduced in decline
depression, =Dopamine
BIPOLAR II Characterized by atleast -psych increased in mania - stable since Reuptake
one episode of major response to there is a high Inhibitors
depressive disorder and trauma leading chance that the
at least one episode of to selective px will get the _______________
hypomania memory same diagnosis Manic and
blocking for the next 5 Hypomanic
years
= atypical
antipsychotics (2nd
generation
CYCLOTHYMIA - 3-5% of the general -related to BPD -sensitive, antipsychotics:
population -biological and hyperactive, and clozapine,
-co-exist with BPD generic data moody during risperidone,
-families with SUD related with childhood olanzapine, and
-ages between 15-25 Mood DO -sx in teens quetiapine)
Emergence of sx _______________
affects school Mood Stabilizers
and socpar
-G = coping strat = Lithium,
Valproic Acid,
MAJOR DEPRESSIVE 5 or more depressive -highest lifetime -triggered by represent a Chronic and Carbamazepine,
DISORDER symptoms during 2-week prevalence with 5- acute stress or change from tends to relapse. Clonazepam
period 17% trauma previous 50% chance of
functioning at recovering after
least one of the the first year.
symptoms
either Persists =
- depressed dysthymic
mood
- loss of
interest or
ppleasure
-Milder form of MDD W= .MC, less than 64 -biological -depressed Before 25 years
-More Chronic -Unmarried, low - psychosocial mood old in 50% of the
-With at least 2 years of income factors -insomnia px
duration -5-6% of the general -poor appetite
population -low energy – Prognosis varies
DYSTHMIA/ fatigue
PERSISTENT -hopelessness 25% do not
DEPRESSIVE DISORDER recover