Examination of the
Cardiovascular System
Erdem ÖZEL,Prof.,MD
Cardiovascular exam
Includes
Appropriate history
Vital Signs: Blood pressure; Pulse: rate, rhythm,
volume
skmattocheck
Assessment distal vasculature (legs, feet,
carotids)vascular disease (atherosclerosis) is a
systemic illness !
examination
Eical
4 basic PE components:
Observation, Palpation, Percussion (omitted in
cardiac exam) & Auscultation
Appropriately/Respectfully
touching your patients
Several Sources of Tension:
beingrespectful is reallyimportant
Area examined reasonably exposed – yet patient
modesty preserved
Palpate sensitive areas to perform accurate exam -
requires touching people w/whom you’ve little
acquaintance – awkward, particularly if opposite
gender
Exam not natural/normal part of interpersonal
interactions - as newcomers to medicine, you’re
particularly aware & hence sensitivea good thing!
Keys To Performing a Respectful &
Effective Exam
Explain what you’re doing (& why) before doing it
Expose minimum amount of skin necessary - “artful” use of
gown & drapes (males & females)
Examining heart & lungs of female patients:
Ask patient to remove bra prior and/or learn to work around bra
Expose side of chest to extent needed
Enlist patient’s assistancepositioning breasts to enable cardiac
exam
Don’t rush
PLEASE... don’t examine body parts thru gown:
Poor technique
You’ll missthings
Observation
Pay attention to:
deformitiesmaybe
Chest shape any
Shortness of breath (@ rest or walking)?
Sitting upright? Able to speak?
? Visible impulse on chest wall from vigorously
contracting ventricle (rare)
hypotrophy
Surface Anatomy
To make good
observation
EE
b
devidest No
I
Finding the sternal manubrium junction
(angle of Louis) important
identify
to
(Key to identfying valve areas)
Valves and Surface Anatomy
left
right
Areas of auscultation correlate w location of each valve
Where you listen will determine what you hear
Palpation-Technique
Fingers across chest, under
breast (explain 1st)
Point of M aximal Impulse
(PMI)apex ventricle that pin-
points w/finger tip; ~70% of
patients - if not palpable, repeat
w/patient on L side
Size of LV – increased
dimension if PMI shifted to L of
mid-clavicular line
Vigor of contraction
Palpable thrill (rare) -
associated w/regurgitant or
stenotic murmurs (feels like
sensation when kink garden
hose)
fromvalves
problems
Palpation-Technique
Right ventricle:
Vigor of contractility
heel of R hand along
sternum
Rarely abnormal with RV
(pulmonary hypertension)
Auscultation-using
part
your
most
important
stethescope
What Are We Listening For ?
Normal valve closure
creates sound
First Heart Sound =s S1
closure of Mitral,
Tricuspid valves
Second Heart Sound =s
S2closure of Pulmonic,
Aortic valves
What Are We Listening For ?
Contraction
Systole =s time between
S1 & S2; Diastole =s time
between S2 & S1 relaxtion
Normally, S1 & S2 =
distinct sounds
Physiologic splitting =s 2
components of second
heart sound (Aortic &
Pulmonic valve closure)
audible w/inspiration
0.5 0.3
diastole systole
Auscultation Technique
Patient lying @ 30-45 degree incline
Chest exposed (male) or loosely fitted gown
(female) • need to see area where placing
stethoscope
Stethoscope must contact skin
Stethoscope w/diaphragm (higher pitched sounds)
engaged
Remember –Don’t examine
thru clothing
Exam options-Female
patients
Auscultation Technique
1. Start over Aortic area2nd Right Intercostal Space
(ICS) – Use Angle of Louis as landmark
intercostalspace
2. Pulmonic area (2nd L ICS)
3. Inch down sternal borderTricuspid area (4th L
ICS)
4. Inch towards Mitral area (4th ICS, mid-clavicular)
5. Listen in ~ 6 places - precise total doesn’t matter
– gives you sense of change In sounds as change
location
Auscultation
In each area, ask yourself:
Do I hear S1? Do I hear S2? Which is louder & what
are relative intensities?
Interval between S1 & S2 (systole) is shorter then
between S2 & S1 (diastole)
Can also determine timing by simultaneously
feeling pulse (a systolic event)
Listen for physiologic splitting of 2nd heart sound
w/inspiration
Murmurs
Murmurs: Sound created by
turbulent flow across valves:
Leakage (regurgitation) when
valve closed
blockage
Obstruction(stenosis) to flow
when normally open
Systolic M urmurs:
Aortic stenosis
Mitral regurgitation
(Pulmonary stenosis, Tricuspid
regurgitation)
Diastolic M urmurs:
TEIFI
Aortic regurgitation
Mitral stenosis
(Pulmonary regurgitation,
Tricuspid stenosis)
stem's
he could ask
important
Murmurs
Characterized by: position in cycle, quality, intensity, location, radiation; can try
to draw it’s shape:
IntensityScale:
1 –barely audible 2- readily audible 3- even louder 4- loud + thrill 5- audible with
only part of
diaphragm on chest 6 – audible w/out stethoscope
Intensity doesn’t necessarily correlate w/severity
Some murmurs best appreciated in certain positions:
Mitral: patient on L side; Aortic: sitting up and leaning forward
• Example – Mitral Regurgitation: H olosystolic, loudest in mitral area, radiates
towards axilla.
51 Physiologic sounds
51
Extra Heart Sounds & S3-S4
Tthologic
Ventricular sounds, occur during diastole
sounds
normal in young patient (~ < 30 yo)
usually LV, rarely RV
S3follows S2
caused by blood from LA colliding w/”left over”
blood in LV • associated w/heart failure.
S4precedes S1
caused during atrial systole
when blood squeezed into non-compliant LV •
associated w/HTN
Extra Heart Sounds & S3-
S4
S3 & S4 are soft, low pitched
Best heart w/bell, laid over LV, w/patient lying on
L side (brings apex of heart closer to chest wall)
Abnormal beyond age ~30
When present, S3 or S4 are referred to as “gallops”
Auscultation- An Ordered
Approach
Do I hear S1? Do I hear S2?
Listen in each major valvular area – think about which sound
should be loudest in each location (S1 loudest region of TV &
MV, S2 loudest AV & PV)
Do I hear physiologic splitting of S2? with inspiration experasion
Do I hear something before S1 (an S4) or after S2 (an S3)?
Do I hear murmur in systole? In diastole?
If a murmur present, note:
intensity,character,duration,radiation
As listen, think about mechanical events that generate the
sounds.
Carotid Arteries
Anatomy
Palpation (ea side
separately!)
Rhythm
Fullness
Auscultation
Radiation of murmurs
? Intrinsic atherosclerosis
– may produce
“shsshhing” noise known
as bruit
Jugular Venous Pressure
(JVP)
Anatomy of Internal
Jugular Vein
Straight line with RA
Manometerreflecting
Central Venous Pressure
(CVP)
JVP Technique
Find correct area – helps to
first identify SCM & triangle
it forms w/clavicle
Look for multi-phasic
pulsations (‘a’, ‘c’ & ‘v’
waves)
Isolate from carotid
pulsations, respirations
Tangential lighting
Hepatojugular reflux (gentle
pressure over liver pushes
blood back into IJ & makes
pulsations more apparent)
JVP Technique
JVP =s 5cm (height sternal
manubrium jxn is above
RA) + vertical distance
from sternal manubrium
jxn to top of pulse wave
Normal < 8 cm
Lower Extremity Vascular Exam-General
observation incl. femoral region
Expose both legs, noting:
asymmetry, muscle
atrophy, joint (knee, ankle)
abnormalities
Focus on Femoral Area:
Inspect - ? Obvious
swellingfemoral hernia v
large lymph nodes (rare)
Palpate lymph nodes
Femoral Region
Identify femoral pulse
Listen over femoral artery
with diaphragm
stethescope for bruits (if
suggestion vascular
disease by hx, exam)
Popliteal Pulse
W/knee slightly bent, push
fingers into popliteal
fossaassess popliteal
artery Relevant if distal
pulses diminished
Vascular Disease of the
Lower Leg
Components:
outflow(arterial)
return(venous,lymphatic)
Clinical Presentations:
Arterial:
pain (supply-demand)
wound healing
RFs for atherosclerosis
Venous:
Edema
Local v systemic etiology
Lymph (relatively uncommon):
Lymphedema:
From obstruction, disruption
Clinical Appearance –Varies with type of
vascular disease
Feet and Ankles
Lower leg & feet @ greatest
risk atherosclerosis (in
particular if vascular dz risk
factors: DM, HTN, Smoking,
Hyperlipidemia, age, known
dz elsewhere)
Observe
? swelling (edema),
discoloration, ulcers, nail
deformities
Look @ bottom of feet,
between toes (problem
areas)
Symmetry?
Feet and Ankles
Palpation
Temperature: Use back of examining hand -
warminflammation; coolatherosclerosis &/or hypo-
perfusion
Capillary refill: push on end of toe or nail bed &
releasecolor returns in < 2-3 seconds;
longeratheroscloerosis &/or hypo-perfusion
Feet and Ankles-Edema
Change in balance of starling
forces (pressures in vessels v
tissues; oncotic forces in vessels v
tissues): Edema
Local leg problems:
Deep Vein Thrombosis
Infection
Trauma
Lymphatic obstruction
Systemic problems:
Heart failure
Pulmonary disease (pulmonary
hypertension, sleep apnea,
thrombosis, etc.)
Kidney disease
Liver disease
Venous stasis
Quantifying edema
A marker of volume status.
Trace (minimal): subtle loss of tendons on top of
foot, contours maleolous
Scales (none validated or compared)
0 to 4+
Depth of pitting left (after applying pressure w/a
finger) in mm
Extent of edema (e.g. limited to feet v up to knee)
Quantifying edema
Dorsalis Pedis Pulse
Palpate Dorsalis Pedis
pulse
Just lateral to extensor
tendon great toe
Use pads of 2-3 fingers of
examining hand
Push gently
If unsure whether feeing
your pulse v patient’s,
measure your carotid or
their radial w/other hand
Graded 0 (not detectable)
to 2+ (normal)
Posterior Tibial Pulse
Palpate Posterior Tibial
Pulse
Located posterior to
medial malleolous
Start on top of malelous &
work towards Achilles
tendon
Use pads of 2-3 fingers,
pushing gently
Same rating scale as for
dorsalis pedis
Summary
Wash hands; gown & drape appropriately
Inspect precordium Palpation of RV and LV; Determination PMI
Auscultation – patient @ 30 degrees
S1 and S2 in 4 valvular areas w/diaphragm
Try to identify physiologic splitting S2
? Murmurs
Assess for extra heart sounds (S3, S4) w/bell over LV
Carotid artery palpation, auscultation
Jugular venous pressure assessment
General lower extremity observation
Assess femoral area (palpation for nodes, pulse); auscultation over femoral artery
Knees – color, swelling; popliteal pulse
Assess ankles/feet (color, temperature, pulses, edema, cap refill