John
John
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
                                                                 Page 1 of 11
                                                                 Final Laboratory Report                          PID     : 5905991
 Differential Counts
  Neutrophil                                            51.3                %                  40 - 75
 Flowcytometry
  Lymphocyte                                            38.0                %                  20 - 45
 Flowcytometry
  Monocytes                                             7.2                 %                  2 - 10
 Flowcytometry
 Absolute Counts
  Absolute Neutrophil Count                             4294                Cells/cmm          2000-7000
 Calculated
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 According to ICSH guideline (international Council for Standardisation in Hematology), the differential counts should be
 reported in absolute numbers.
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                                                Dr.Selvi R
                                                                                                     Consultant Biochemist
 Verified by                                                               Page 4 of 11                                                           MC-5972
                                                         Final Laboratory Report                      PID     : 5905991
INTERPRETATIONS
     •   Circulating TSH measurement has been used for screening for euthyroidism, screening and diagnosis for
         hyperthyroidism & hypothyroidism. Suppressed TSH (<0.01 µIU/mL) suggests a diagnosis of hyperthyroidism
         and elevated concentration (>7 µIU/mL) suggest hypothyroidism. TSH levels may be affected by acute illness
         and several medications including dopamine and glucocorticoids. Decreased (low or undetectable) in Graves
         disease. Increased in TSH secreting pituitary adenoma (secondary hyperthyroidism), PRTH and in
         hypothalamic disease thyrotropin (tertiary hyperthyroidism). Elevated in hypothyroidism (along with decreased
         T4) except for pituitary & hypothalamic disease.
     • Mild to modest elevations in patient with normal T3 & T4 levels indicates impaired thyroid hormone reserves &
         incipent hypothyroidism (subclinical hypothyroidism).
     • Mild to modest decrease with normal T3 & T4 indicates subclinical hyperthyroidism.
     • Degree of TSH suppression does not reflect the severity of hyperthyroidism, therefore, measurement of free
         thyroid hormone levels is required in patient with a supressed TSH level.
  CAUTIONS
  Sick, hospitalized patients may have falsely low or transiently elevated thyroid stimulating hormone.
  Some patients who have been exposed to animal antigens, either in the environment or as part of treatment or
  imaging procedure, may have circulating antianimal antibodies present. These antibodies may interfere with the
  assay reagents to produce unreliable results.
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                         Dr.Selvi R
                                                                              Consultant Biochemist
 Verified by                                               Page 5 of 11                                           MC-5972
                                                            Final Laboratory Report                          PID     : 5905991
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                              Dr.Selvi R
                                                                                   Consultant Biochemist
 Verified by                                                    Page 6 of 11                                             MC-5972
                                                                Final Laboratory Report                        PID      : 5905991
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                                  Dr.Selvi R
                                                                                       Consultant Biochemist
 Verified by                                                     Page 7 of 11                                               MC-5972
                                                                 Final Laboratory Report                           PID     : 5905991
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                                    Dr.Selvi R
                                                                                         Consultant Biochemist
 Verified by                                                       Page 8 of 11                                                MC-5972
                                                                     Final Laboratory Report                                PID        : 5905991
  INTERPRETATION
     • Levels <10 ng/mL may be associated with more severe abnormalities and can lead to inadequate mineralization of newly formed osteoid, resulting
        in rickets in children and osteomalacia in adults. In these individuals, serum calcium levels may be marginally low, and parathyroid hormone (PTH)
        and serum alkaline phosphatase are usually elevated. Definitive diagnosis rests on the typical radiographic findings or bone
        biopsy/histomorphometry.
     • Patients who present with hypercalcemia, hyperphosphatemia, and low PTH may suffer either from ectopic, unregulated conversion of 25-OH-VitD
        to 1,25 (OH)2-VitD, as can occur in granulomatous diseases, particularly sarcoidosis, or from nutritionally-induced hypervitaminosis D. Serum 1,25
        (OH)2-VitD levels will be high in both groups, but only patients with hypervitaminosis D will have serum 25-OH-VitD concentrations of >80 ng/mL,
        typically >150 ng/mL.
     • Patients with CKD have an exceptionally high rate of severe vitamin D deficiency that is further exacerbated by the reduced ability to convert 25-
        OH- VitD into the active form, 1,25 (OH)2-VitD. Emerging evidence also suggests that the progression of CKD & many of the cardiovascular
        complications may be linked to hypovitaminosis D.
     • Approximately half of Stage 2 and 3 CKD patients are nutritional vitamin D deficient (25-OH-VitD, less than 30 ng/mL), and this deficiency is more
        common among stage 4 CKD patients. Additionally, calcitriol (1,25 (OH)2-VitD) levels are also overtly low (less than 22 pg/mL) in CKD patients.
        Similarly, vast majority of dialysis patients are found to be deficient in nutritional vitamin D and have low calcitriol levels. Recent data suggest an
        elevated PTH is a poor indicator of deficiencies of nutritional vitamin D and calcitriol in CKD patients.CAUTIONS Long term use of anticonvulsant
        medications may result in vitamin D deficiency that could lead to bone disease; the anticonvulsants most implicated are phenytoin, phenobarbital,
        carbamazepine, and valproic acid.
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)
 AUTO                                                                                           Dr.Selvi R
                                                                                                Consultant Biochemist
 Verified by                                                           Page 9 of 11                                                           MC-5972
                                                                 Final Laboratory Report                           PID     : 5905991
  pH                                                    5.0                                     5-9
 Indicator
 Chemical Examination
  Protein                                               Negative            mg/dL               Negative
 Reflectance Photometry (Protein Error of
 Principle indicator)
 Microscopic Examination
  Red Blood Cell                                        0.0                 /HPF                0-2.3 cells/hpf
  Pus Cells                                             2.9                 /HPF                0-2.7 cells/hpf
  Epithelial Cell                                       0.0                 /HPF                0-1.1 cells/hpf
  Cast                                                  0.0                 /HPF                0-0.5 p/hpf
  Pathological Cast                                     0.0                 /HPF                NIL
 Reflectance Photometry
 Crystals
  Calcium oxalate Monohydrate                           1.5                 /HPF                Nil
  Calcium oxalate Dihydrate                             0.0                 /HPF                Nil
  Triple phosphate                                      0.0                 /HPF                Nil
  Uric Acid                                        H 28.5                   /HPF                0-1.4 p/hpf
 Phase Contrast Microscopy (Sedimentation
 based with AIEM)
 # For test performed on specimens received or collected from non-NDPL locations, it is presumed that the specimen belongs to the patient named or identified as labeled
 on the container/test request and such verification has been carried out at the point generation of the said specimen by the sender. NDPL will be responsible Only for the
 analytical part of test carried out. All other responsibility will be of referring Laboratory.
Note:(LL-VeryLow,L-Low,H-High,HH-VeryHigh,A-Abnormal)