Abnormal Psy
Abnormal Psy
ADHD
ADHD (Attention-Deficit/Hyperactivity Disorder) is a neurodevelopmental disorder that
primarily affects children but can continue into adulthood. It is characterized by symptoms such
as inattention, hyperactivity, and impulsiveness
Earlier as ADD or Attention-Deficit Disorder, is an older term that was used to describe
individuals who had problems with attention but did not display significant hyperactivity or
impulsiveness. ADD is now considered part of the inattentive subtype of ADHD. The term was
officially replaced by ADHD in the 1994 revision of the DSM (Diagnostic and Statistical Manual
of Mental Disorders), and now ADHD encompasses both inattentive and hyperactive-impulsive
symptoms.
The diagnostic criteria for ADHD (Attention-Deficit/Hyperactivity Disorder) are outlined in the
Diagnostic and Statistical Manual of Mental Disorders, 5th edition (DSM-5), published by the
American Psychiatric Association. These criteria help professionals accurately diagnose ADHD
by assessing the presence and impact of symptoms in children or adults.
1. Onset of Symptoms
• ADHD symptoms must present during the developmental period, typically before the age
of 12. Although symptoms often appear earlier (in preschool years), they should be
noticeable before adolescence to be considered part of ADHD diagnosis.
• Symptoms must be observed in two or more settings (e.g., home, school, social
interactions). This is important because ADHD is a pervasive disorder that affects various
aspects of a child’s life, not just one environment.
• Example: A child may have difficulty concentrating both in class and while doing
homework, as well as when playing with friends.
3. Duration of Symptoms
• Symptoms must have persisted for at least 6 months. These behaviors should be
inappropriate for the child’s developmental level.
• Academic Milestones: Difficulty learning to read, write, or complete tasks like other
children their age.
• Example: A child might struggle to learn to read because they can’t focus on the text long
enough.
• Social Milestones: Children may experience difficulties in understanding how to take
turns, share, or engage in reciprocal play.
• Example: A child might have trouble waiting for their turn during games, leading to
conflicts with peers.
• Occupational Milestones (older children/adolescents): Difficulty in completing tasks at
part-time jobs, managing schedules, or taking responsibility for assigned tasks.
• Example: An older child might lose track of shifts or miss deadlines at their job.
Conclusion
To diagnose ADHD, healthcare providers look for persistent and impairing symptoms of
inattention or hyperactivity-impulsivity that start before the age of 12, affect multiple areas of the
child’s life, and last for at least six months. These symptoms must not be better explained by
another condition and should result in functional impairment in personal, academic, or social areas.
Early diagnosis and intervention can help manage ADHD effectively, improving the child’s quality of
life.
3. Non-Pharmacological Interventions:
Example: A 7-year-old child with ADHD might have difficulty sitting still and focusing in class. In
behavioral therapy, the therapist might use a reward system where the child earns a sticker for
every 15 minutes they remain seated and attentive. After earning a certain number of stickers,
the child can choose a reward, such as extra playtime or a small treat. Over time, this positive
reinforcement helps the child develop better self-regulation and focus.
CBT helps individuals with ADHD become more aware of how their thoughts affect their
behavior. This therapy focuses on changing negative thought patterns that can contribute to
impulsive or inattentive behavior.
Example: A teenager with ADHD might struggle with procrastination, particularly on school
assignments. In CBT, the therapist would help the teen identify the negative thoughts that
contribute to procrastination, such as “I’ll never be able to finish this, so why start?” The
therapist might teach the teen how to break assignments into smaller, manageable tasks and set
achievable goals to help them stay on track.
This intervention involves teaching parents strategies to manage their child’s behavior more
effectively. It focuses on creating structure, establishing clear expectations, and using consistent
consequences and rewards.
Example: A parent might struggle with their child’s constant forgetfulness and inability to
complete chores. During parent training, the therapist might suggest setting up a chore chart
with clear instructions and deadlines for each task. The parent is encouraged to use a token
system where the child earns tokens for completing tasks, which can be exchanged for
privileges, like screen time. The parent is also trained to give consistent and calm consequences
when the child does not complete tasks.
4. School-Based Interventions:
School accommodations can help children with ADHD perform better academically and socially.
Teachers can implement strategies to provide structure and minimize distractions in the
classroom.
Example: A child with ADHD who has trouble focusing on tasks might benefit from a 504 plan or
an Individualized Education Program (IEP). This might include seating the child near the front
of the class, providing extra time for tests, or giving the child frequent breaks to move around.
Teachers might also use a visual schedule that outlines daily activities to help the child stay
organized and reduce anxiety about transitions
Children and teens with ADHD often struggle with social interactions. Social skills training helps
them learn appropriate ways to interact with peers, such as waiting their turn to speak,
maintaining conversations, and understanding social cues
Example: A child with ADHD might frequently interrupt others during group activities, leading
to difficulties forming friendships. In social skills training, the child is taught to recognize when
it’s their turn to speak by practicing with role-playing activities. They might also learn
techniques for managing impulsive behaviors, such as counting to five before responding to
someone.
Physical exercise and mindfulness can be beneficial in reducing hyperactivity and improving
focus. Research shows that regular physical activity can help manage ADHD symptoms.
Conclusion:
ADHD management often requires a combination of both pharmacological (e.g., stimulant and
non-stimulant medications) and non-pharmacological interventions (e.g., psychoeducation,
therapy, coaching, and accommodations). A comprehensive and tailored Interventions for
ADHD (Attention-Deficit/Hyperactivity Disorder) are typically multi-faceted, addressing
behavioral, cognitive, and sometimes medical aspects of the disorder. The goal is to help
individuals manage their symptoms and improve their functioning in various areas of life, such
as at school, home, or work. Below are some key interventions with examples:
Causes
Genetic Factors
Family History: ADHD tends to run in families, suggesting a genetic component. Studies
show that individuals with a parent or sibling with ADHD are more likely to develop the
disorder.
2 . Brain Structure and Function
Neurotransmitter Imbalances: Research indicates that imbalances in neurotransmitters,
particularly dopamine and norepinephrine, may play a role in ADHD.
Brain Structure: Imaging studies have found differences in the size and activity of certain
brain areas, such as the prefrontal cortex, which is involved in attention and impulse
control.
Barkley’s theory (1997) highlights that the core issue in ADHD is the inability to inhibit
responses due to impairments in the prefrontal cortex. This impairment affects:
1. Inhibition of responses
2. Mental representation and memory
3. Motivation and emotional regulation
4. Reconstruction of thoughts and actions
3. Environmental Factors
Prenatal Exposure: Exposure to substances during pregnancy, such as tobacco smoke,
alcohol, or drugs, can increase the risk of ADHD in children.
Lead Exposure: Exposure to lead and other environmental toxins, particularly in early
childhood, has been linked to increased ADHD symptoms.
4. Developmental Factors
Premature Birth: Children born prematurely or with low birth weight may be at a higher
risk for developing ADHD.
Early Childhood Trauma: Adverse experiences in early childhood, such as abuse, neglect,
or significant stress, can contribute to the development of ADHD symptoms.
ADHD in Girls:
ADHD in girls is often missed or misdiagnosed because their symptoms tend to be less obvious than
boys’. Girls often internalize their symptoms, which can lead to anxiety and low self-esteem.
Educators and clinicians may overlook these subtler symptoms.
ADHD in Adulthood:
Adult ADHD manifests primarily as difficulties with attention and organization. Adults with ADHD
tend to make careless mistakes, lose things, and have issues prioritizing tasks due to deficits in
executive functioning. These challenges can affect relationships, both personal and professional.
Comorbidity refers to the presence of one or more additional disorders co-occurring with a
primary disorder. In the case of ADHD, it is common for individuals to experience other conditions
alongside it. Here are some of the most frequent comorbidities associated with ADHD:
Substance Use Disorders
Example: Adolescents and adults with ADHD are at a higher risk for developing substance
use disorders. A young adult with untreated ADHD might turn to alcohol or drugs as a way
to cope with their symptoms, leading to addiction issues.
Anxiety Disorders
Example: Many children and adults with ADHD also struggle with anxiety disorders, such
as generalized anxiety disorder or social anxiety disorder. For instance, a child with ADHD
may feel overwhelmed in social situations, leading to heightened anxiety and avoidance
behaviors.
2. Depressive Disorders
Example: Depression can often co-occur with ADHD. An adolescent with ADHD may
experience feelings of low self-esteem or frustration due to their challenges with attention
and impulse control, leading to depressive symptoms.
3. Learning Disabilities
Example: Children with ADHD frequently have learning disabilities, such as dyslexia or
dyscalculia. For example, a child with ADHD might struggle with reading comprehension
due to distractibility, which can be compounded by dyslexia, making it even harder for them
to succeed academically.
Autism Spectrum Disorder (ASD) is classified in the DSM-5-TR (Diagnostic and Statistical Manual of
Mental Disorders). It includes impairments in social communication, restricted and repetitive
behaviors, and interests. The severity and persistence of the condition vary, but improvement is
possible with appropriate support.
The Centers for Disease Control and Prevention (CDC) reported in 2007 that approximately 1 out of
every 150 children in the United States was diagnosed with autism spectrum disorder. Over the
years, this rate has increased significantly to around 1 in 54 children. This rise is likely due to
changes in diagnostic criteria, greater awareness, and improved reporting practices.
Example: A 2007 study might have identified fewer children with autism due to less awareness or
different diagnostic tools compared to a more recent study that identifies more children because of
better diagnostic standards.
The diagnosis of ASD in DSM-5-TR replaces earlier terms like autistic disorder and
Asperger’s disorder. ASD now exists on a continuum of severity, rather than as separate disorders.
This shift helps to better distinguish children with typical development from those with ASD, while
also accounting for a broad range of behaviors and impairments.
3. Clinicians assess ASD in two main areas:
• Social communication and relationships
• Restricted, repetitive patterns of behavior or interests
4. Children with ASD might exhibit delayed language development, difficulty engaging
in social interactions, and atypical body language (e.g., avoiding eye contact or showing unusual
facial expressions). These behaviors may seem odd or unusual to others.
5. Risk of Self-Harm:
Definition: Individuals with ASD are at a higher risk of self-harm, particularly by the age of 16. This
is possibly due to the combination of social challenges, sensory issues, and difficulties in emotional
regulation.
Example: Some teens with ASD might engage in self-harming behaviors like hitting themselves or
biting when they feel overwhelmed by sensory stimuli or social stress.
8. Regressive Autism:
Definition: Some children with ASD develop typically for the first few years but then lose language
and motor skills around the age of 10. This is known as regressive autism.
Example: A child may start speaking and interacting typically, but around age 3 or 4, they might
stop talking and lose skills they had previously developed, such as riding a bike or using utensils.
Onset in the Developmental Period according to dsm5
Early Signs: Symptoms of ASD typically manifest in the early developmental period, often
before age 3. However, they may not become fully apparent until social demands exceed the
individual's limited capacities.
Example of Symptoms: Early signs can include delays in communication (e.g., lack of
babbling or gestures), social engagement (e.g., not responding to name), or repetitive
behaviors.
Impairments in Key Areas
1. Personal Functioning:
o Self-Care Skills: Individuals may show delays in self-care milestones (e.g., dressing,
feeding) and may need support for routine tasks.
o Independence: Challenges in developing self-management skills can impact daily
living.
2. Social Functioning:
o Social Interaction Difficulties: Individuals often have difficulty understanding
social cues and engaging in reciprocal conversations. They may not share interests
or emotions effectively.
o Friendship Challenges: There is a marked impairment in the ability to form and
maintain friendships, leading to social isolation.
3. Academic Functioning:
o Learning Differences: Academic performance can be affected by difficulties with
communication, attention, and social interactions. Some children may have specific
learning disabilities or require individualized education plans (IEPs).
o Classroom Engagement: Challenges with group work or participating in classroom
discussions can hinder academic progress.
4. Occupational Functioning:
o Transition to Adulthood: As individuals age, they may struggle with employment
due to social difficulties, understanding workplace norms, and managing tasks.
o Skill Development: Difficulties in acquiring skills necessary for independent living
(e.g., time management, budgeting) can affect occupational success.
Delays in Achieving Milestones
Communication: Delays may manifest as limited spoken language, use of echolalia, or
challenges in initiating and sustaining conversations.
Motor Skills: Fine and gross motor skills may be delayed, impacting physical activities and
tasks requiring coordination.
Social Play: Engaging in imaginative or cooperative play with peers can be limited, affecting
social development and the ability to form friendships.
Causes
Causes
1. Genetic Factors
Hereditary Influence: Autism tends to run in families, suggesting a genetic component.
Specific genes have been implicated, including those related to brain development and
function.
Chromosomal Abnormalities: Some studies have identified chromosomal abnormalities
that may increase the risk of autism, such as variations in chromosomes 7, 15, and 16.
2. Environmental Factors
Prenatal Exposure: Factors during pregnancy, such as maternal infections (e.g., rubella),
exposure to toxins (e.g., pesticides, heavy metals), or medications (e.g., valproic acid), can
increase the risk of autism.
Birth Complications: Low birth weight, prematurity, or complications during delivery may
be associated with a higher risk of autism.
3. Neurological Factors
Brain Structure and Function: Research has shown differences in the brain structure and
connectivity in individuals with autism, particularly in areas responsible for social behavior
and communication.
Neurotransmitter Imbalances: Abnormal levels of neurotransmitters like serotonin and
dopamine may play a role in the development of autism.
4. Metabolic and Immunological Factors
Immune System Function: Some studies suggest that immune system abnormalities may
contribute to the development of autism, particularly if the immune system reacts
abnormally to environmental exposures.
Metabolic Disorders: Certain metabolic conditions, such as phenylketonuria (PKU), can
lead to neurodevelopmental issues, including symptoms resembling autism.
5. Sociocultural Factors
Parental Age: Advanced parental age at the time of conception has been linked to an
increased risk of autism, although the mechanisms are not fully understood.
Socioeconomic Factors: While not a direct cause, lower socioeconomic status can affect
access to healthcare and early intervention, impacting developmental outcomes.
Rett Syndrome:
Overview: Rett syndrome is a genetic neurological disorder that becomes noticeable within
the first 18 months of life and primarily affects girls.
• Symptoms:
• Constant repetitive hand movements (such as wringing or clapping).
• Loss of speech and purposeful hand skills.
• Gait abnormalities (walking problems).
• Complete or partial loss of social interaction skills.
• Progression:
As children age, social engagement deteriorates into what is known as regressive Rett
syndrome. Early symptoms may be confused with autism, but DSM-5-TR advises that Rett
syndrome should be used in differential diagnoses of ASD when children regress after a
period of normal development.
• Genetics and Treatment:
• Caused by mutations in the MECP2 gene, which disrupts normal brain functioning.
• Researchers have identified potential mouse model studies to explore treatment, but no
effective clinical therapies currently exist. However, behavioral and rehabilitation
interventions focusing on motor, communication, and cognitive skills have shown some
promise.
• Psychological interventions are also noted as useful in managing symptoms of Rett
syndrome.
Before World War II, mental disorders were predominantly understood through a physiological
lens, with a strong emphasis on biological causes. Psychological conditions were often thought to
stem from physical ailments or abnormalities in the brain, neglecting environmental or social
factors.
Historical Perspective on Mental Disorders
1. Physiological Understanding:
o In the early 19th century, mental disorders were largely considered the result of
physiological issues. Conditions that we now recognize as Post-Traumatic Stress
Disorder (PTSD) were often labeled as "nervous disorders" or "hysteria," focusing
primarily on physical symptoms.
o Treatments were primarily medical, including rest cures, hydrotherapy, and
occasionally more invasive procedures, without addressing psychological or
contextual factors.
2. Shell Shock in World War I:
o The term "shell shock" emerged during World War I to describe soldiers who
experienced debilitating psychological symptoms following combat. Initially, these
symptoms were thought to result from physical injuries, such as concussions from
blasts.
o As understanding evolved, it became evident that many soldiers who had not
sustained any physical injuries still suffered severe psychological distress, indicating
a psychological response to the traumas of war.
3. Post-World War II Insights:
o After World War II, further research and clinical observations revealed that PTSD
could affect individuals regardless of physical injuries. This shift in understanding
highlighted the importance of psychological and environmental factors, including
the impact of trauma exposure and stress.
o The recognition that healthy individuals could develop PTSD as a result of traumatic
experiences led to a more comprehensive understanding of mental health,
incorporating the interplay between biological, psychological, and environmental
influences.
Conclusion
The evolution of thought regarding mental disorders, particularly PTSD, illustrates a significant
shift from a purely physiological perspective to a more holistic understanding that includes
psychological and environmental factors. This change has been crucial for developing effective
treatments and support systems for individuals coping with trauma and mental health challenges.
DSM 1 – 1952
The DSM-I (Diagnostic and Statistical Manual of Mental Disorders, First Edition) was published in
1952 by the American Psychiatric Association, featuring 106 reaction. Prior to this publication,
mental health conditions were not categorized as "disorders," reflecting a less standardized
understanding of mental health.
Historical Context and Features of DSM-I
1. Psychoanalytic Influence:
o The DSM-I was significantly influenced by the psychoanalytic approach, particularly
the work of Adolf Meyer, who emphasized a psychodynamic perspective. This
approach focused on unconscious processes and childhood experiences as critical
factors in understanding mental health.
2. Terminology and Clarity:
o The language used in the DSM-I was often wordy and complex, lacking clarity.
Descriptions of disorders were lengthy and sometimes vague, making it difficult for
clinicians to identify specific diagnostic criteria for particular conditions.
3. Classification of Disorders:
o Disorders in DSM-I were categorized primarily based on perceived causality,
dividing them into two main groups:
Organic Disorders: Linked to identifiable impairments in brain tissue or
neurological functions.
Psychogenic Disorders: Considered to arise from psychological or
emotional factors, reflecting the belief in the mind's role in mental health.
DSM 2 – 1968
Increased Number of Diagnoses:
The DSM-II expanded its scope to include approximately 182 diagnoses, reflecting a broader
understanding of mental health conditions.
Terminology Changes:
The terminology used in DSM-I, which often referred to psychoanalysis, was largely
dropped. This shift signified a move toward more descriptive and clinically relevant
language.
Psycho-Physical Disorders:
While the focus on psychoanalytic terminology diminished, psycho-physical disorders
remained in the classification. These disorders were viewed as having both psychological
and physical components.
Removal of Homosexuality as a Disorder:
In 1974, following significant political and social advocacy, homosexuality was removed
from the DSM-II as a mental disorder. This change reflected a growing understanding of
sexual orientation and a movement toward reducing stigma.
Ego-Dystonic Homosexuality:
Despite the removal of homosexuality as a disorder, the concept of ego-dystonic
homosexuality (where an individual's sexual orientation is in conflict with their personal
identity) remained in the manual until the DSM-III
DSM-III (1980)
The DSM-III, published in 1980, marked a significant transformation in the field of psychiatry,
reflecting a shift toward a more empirical and descriptive approach to mental disorders. Here are
the key features and changes:
1. Expanded Diagnoses:
o The DSM-III included approximately 265 disorders, significantly broadening the
scope of psychiatric diagnosis.
2. Shift from Psychodynamic to Empirical Perspectives:
o This edition dropped the psychoanalytic perspective that characterized earlier
versions, favoring an empirically based approach. It emphasized observable
symptoms and standardized diagnostic criteria.
3. Influence of Emil Kraepelin:
o The DSM-III drew heavily on the work of German psychiatrist Emil Kraepelin,
particularly his concepts regarding the classification of mental disorders. Kraepelin
emphasized the biological and genetic underpinnings of conditions such as
"dementia praecox," which was later renamed schizophrenia.
4. Introduction of Pharmacopsychology:
o Kraepelin also contributed to the field of pharmacopsychology, recognizing the
impact of biological factors on mental health and the potential for pharmacological
treatments.
5. Emergence of a New Theoretical Orientation:
o In the 1960s, a group of psychiatrists at Washington University, dissatisfied with the
psychoanalytic emphasis of previous DSM editions, sought to return psychiatry to
its medical roots. This movement advocated for a focus on descriptive and
epidemiological approaches in psychology.
6. Reliability and Validity:
o The changes implemented in DSM-III resulted in a manual that was much more
reliable and valid than its predecessors, establishing a clearer foundation for
diagnosing mental disorders.
DSM-III-R Overview
Year Published: 1987
Number of Disorders: Approximately 292
Key Features
1. Removal of the Neurological Exclusion Rule:
o The DSM-III-R continued the trend established by DSM-III by eliminating the
neurological exclusion rule, which previously restricted the diagnosis of certain
mental disorders in the presence of neurological conditions. This change allowed for
a more nuanced understanding of mental health, acknowledging that mental
disorders could coexist with neurological issues.
2. Increased Recognition of Comorbidity:
o As a result of the removal of this exclusion rule, comorbidity—where an individual
has multiple co-occurring mental disorders—was more widely recognized. This
acknowledgment improved clinical practice by allowing for more comprehensive
assessments and treatment plans.
3. Refinement of Diagnostic Criteria:
o DSM-III-R refined diagnostic criteria for various disorders, enhancing clarity and
precision in diagnosis. This revision aimed to improve the reliability of diagnoses
and better reflect the complexity of mental health conditions.
DSM-IV Overview
Year Published: 1994
Number of Disorders: Approximately 365
Key Improvements
1. Comprehensive Diagnostic Criteria:
o DSM-IV provided more detailed and specific diagnostic criteria for each disorder,
improving clarity and consistency in diagnoses. This helped clinicians better
understand the nuances of various mental health conditions.
2. Multiaxial System Enhancement:
o The multiaxial classification system was retained and further refined. Each axis was
clearly defined, allowing for a more holistic assessment of the individual, including
clinical disorders, personality disorders, medical conditions, psychosocial factors,
and overall functioning.
3. Incorporation of New Disorders:
o DSM-IV introduced several new disorders based on emerging research and clinical
observations, expanding the classification to better capture the diversity of mental
health issues.
1. Elimination of the Multi-Axial System: The traditional five-axis system used in the
DSM-IV was removed. Diagnoses now focus on a single-axis approach, where all mental and medical
conditions are considered together without separate axes.
2. New Disorders: A few new disorders were introduced, such as:
• Hoarding Disorder: Previously considered a subtype of obsessive-compulsive
disorder (OCD), it was recognized as a distinct condition.
• Binge Eating Disorder: Elevated from a condition under “eating disorders not
otherwise specified” (EDNOS) to its own full diagnosis.
• Disruptive Mood Dysregulation Disorder (DMDD): Created to address the over-
diagnosis of pediatric bipolar disorder, this condition is characterized by chronic irritability and
frequent temper outbursts in children.
• Premenstrual Dysphoric Disorder (PMDD): Moved from an appendix of conditions
needing further study to a formal diagnosis.
3. Reorganization of Categories:
• Mood Disorders were divided into two separate categories:
• Depressive Disorders: Includes conditions like major depressive disorder.
• Bipolar and Related Disorders: Separates bipolar disorders into their own category
to distinguish them more clearly from unipolar depression.
4. Removal of the Bereavement Exclusion: In earlier versions of the DSM, individuals
experiencing symptoms of depression after the death of a loved one were excluded from a diagnosis
of major depressive disorder (MDD) for two months, known as the bereavement exclusion. The
DSM-5 removed this exclusion, allowing for the diagnosis of MDD even if symptoms appear shortly
after a significant loss, acknowledging that grief and depression can coexist.
DSM-5-TR Overview
The DSM-5 Text Revision (DSM-5-TR) was published in 2021, updating the original DSM-5 from
2013.
Major Changes
1. New Diagnoses Added:
o Prolonged Grief Disorder
3. Cultural Updates:
o Emphasis on addressing race and discrimination in diagnoses.
Specific Updates
Prolonged Grief Disorder: Recognizes persistent grief symptoms affecting daily
functioning.
Unspecified Mood Disorder: Reinstated for mood symptoms that don’t meet full criteria.
Stimulant-Induced Mild Neurocognitive Disorder: Addresses cognitive effects from
stimulant use.
No suicidal Self-Injury (NSSI): Defined as self-harm without suicidal intent, allowing for
targeted clinical attention.
Attenuated Psychosis Syndrome (APS): Captures subclinical psychotic symptoms to
prevent misdiagnosis.
This section discusses Specific Learning Disorders (SLDs), which are characterized by delays or
deficits in acquiring basic academic skills like reading, writing, or mathematics, despite normal
intelligence levels.
1. General Definition:
• Children with SLDs struggle to develop skills that are expected at their age,
education, and intelligence levels. Their performance is lower than what would be expected.
• For example, an 8-year-old child may still be unable to read simple words or solve
basic math problems that other children of the same age can.
1. Language Disorder:
• Children with a language disorder have trouble expressing themselves in ways that
are suitable for their age or developmental level. They may use simplified grammar, leave out key
words, or use words in the wrong order.
• Example: A child with this disorder might say, “I go park yesterday” instead of “I
went to the park yesterday,” showing difficulty with verb tense.
• In this condition, the child has trouble with speech sounds, often substituting,
omitting, or mispronouncing them. This affects how clearly they speak.
• Example: A child may replace the “k” sound with a “t” sound, saying “tiss” instead of
“kiss.” While some speech errors are cute in young children, they may persist and cause problems
in school.
• Children who stutter have disruptions in their speech flow, such as repeating
sounds or prolonging syllables. They may also avoid difficult words or have tense speech.
• Example: A child might struggle to say a word, starting with “s-s-s-sun” instead of
“sun,” or use fillers like “um” to delay speech.
• Impact: Research has shown a connection between stuttering and negative
academic outcomes, partly because children may become anxious about speaking and be less
engaged in class.
• Intervention: Treatment includes not only speech therapy but also building
resilience. Parents can help by creating supportive environments where the child can practice skills,
such as dressing independently or solving puzzles, to boost their confidence.
According to the DSM-5 (Diagnostic and Statistical Manual of Mental Disorders, 5th Edition),
the diagnostic criteria for Speech Sound Disorder (previously referred to as phonological disorder
or articulation disorder) and Specific Learning Disorder related to speech include the following:
In the context of learning disorders where speech-related issues might arise, the DSM-5 specifies
criteria under Specific Learning Disorder:
1. Difficulties learning and using academic skills, as indicated by at least one of the
following symptoms that have persisted for at least 6 months, despite the provision of interventions
that target those difficulties:
• Inaccurate or slow and effortful word reading (commonly associated with dyslexia).
• Difficulty understanding the meaning of what is read.
• Spelling difficulties.
• Difficulties with written expression (e.g., grammatical or punctuation errors, poor
paragraph organization).
2. The affected academic skills are substantially below those expected for the
individual’s age and cause significant interference with academic or occupational performance, or
with activities of daily living.
3. The learning difficulties began during the school-age years but may not become fully
evident until the demands for those affected academic skills exceed the individual’s capacities.
4. The learning difficulties are not better accounted for by intellectual disabilities,
uncorrected visual or auditory problems, other mental or neurological disorders, or psychosocial
adversity.
Exclusions for Speech Sound Disorder or Learning Disorder:
• The issue is not due to sensory deficits (like hearing impairment), neurological conditions,
or intellectual disabilities.
• Other environmental factors such as lack of instruction or socio-cultural differences are also
ruled out before diagnosis.
Causes
1. Genetic Factors: Family history of speech and language disorders.
2. Neurological Conditions: Issues with brain development or injury affecting
communication.
3. Hearing Loss: Difficulty hearing can impede language development.
4. Environmental Factors: Lack of exposure to language-rich environments during critical
developmental periods.
5. Psychological Factors: Anxiety or trauma may affect speech fluency and confidence.
6. Developmental Disorders: Conditions like autism spectrum disorder can impact
communication skills
7. Intervention Strategies:
• The best approach to address SLDs is early intervention, using evidence-based
practices like the Response to Intervention (RTI) model. Children are given targeted interventions
and their progress is monitored.
• Example: A child who struggles with reading might receive extra support through
special reading programs in school. If they don’t respond to this, they may undergo further
assessment to identify more specific interventions.
8. Vocational Support for Adults:
• Adults with learning disorders can benefit from vocational rehabilitation programs,
where they receive training tailored to their needs.
• Example: An adult with dyslexia may receive job coaching to help them use tools like
text-to-speech software, making it easier to perform work tasks.
For Speech Sound Disorder, signs usually appear around ages 3 to 4, and if difficulties persist
beyond age 7 to 8, it may indicate a disorder.
For Specific Learning Disorder, it typically becomes noticeable around ages 6 to 7 when
children begin formal education.
Motor Disorder - A motor disorder is a condition that impairs a person's ability to control and
coordinate movements. It can manifest as difficulties with fine or gross motor skills, involuntary
movements, or abnormal muscle tone.
Causes
Genetic factors
Neurological differences
Environmental influences (e.g., limited opportunities for physical activity)
Interventions
Occupational Therapy: Focused on improving motor skills through tailored activities (e.g.,
sports, crafts).
Physical Therapy: Helps improve balance and coordination through specific exercises.
Examples: Practicing specific tasks, using adaptive equipment, and engaging in group
activities to promote social skills.
3. Tic Disorders
Definition
Tic disorders involve sudden, rapid, recurrent motor movements or vocalizations that are
involuntary.
DSM-5 Diagnostic Criteria
A. The presence of multiple motor tics and one or more vocal tics over a period.
B. The tics occur many times a day, nearly every day, or intermittently over a period of
more than one year.
C. The tics cause significant distress or impairment in social, occupational, or other areas of
functioning.
D. The onset is before age 18, and the tics are not attributable to substance use or another
medical condition.
Age of Onset: Tic disorders often begin in early childhood, typically between ages 5 and 10. The
onset can be sudden or gradual, with motor tics often appearing before vocal tics. Tics may
fluctuate in severity and can change in type over time.
Causes
Genetic predisposition
Environmental factors (e.g., stress, infections)
Neurological factors
Interventions
Cognitive Behavioral Therapy (CBT): Often includes habit reversal training.
Medications: Such as antipsychotics or other medications to help manage tics.
Examples: Behavioral strategies to manage tics in social situations, family education, and
support.
Tourette Syndrome is a complex condition involving involuntary movements and sounds known
as tics. Tics are sudden, rapid, recurrent movements (motor tics) or vocalizations (vocal tics).
Symptoms
1. Motor Tics: These can include:
o Blinking
o Head jerking
o Shoulder shrugging
o Facial grimacing
o Throat clearing
Intellectual Disability
Definition: Intellectual disability is characterized by limitations in intellectual functioning and
adaptive behavior, affecting everyday functioning and development.
Diagnostic Criteria (DSM-5)
1. Deficits in Intellectual Functioning:
o Significantly below average intellectual functioning, typically an IQ score of around
70 or below (considering a standard deviation of 15).
2. Deficits in Adaptive Functioning:
o Impairments in adaptive functioning in one or more areas, such as:
Intellectual disability is categorized into four levels of severity based on the degree of impairment
in intellectual functioning and adaptive behavior. Here’s a brief overview of each level:
1. Mild
Intellectual Functioning: IQ between 50-70.
Adaptive Functioning: Individuals may have difficulty with complex tasks but can often
live independently with minimal support. They may require assistance with budgeting, job
skills, and understanding social cues.
Development: May achieve academic skills up to about a 6th-grade level. Many can hold
jobs and manage daily activities.
2. Moderate
Intellectual Functioning: IQ between 35-49.
Adaptive Functioning: Individuals require more support in daily living, such as personal
care and communication. They often need supervision in social situations and may work in
supervised settings.
Development: Typically achieve academic skills up to about a 2nd-grade level. They can
learn basic self-care skills with guidance.
3. Severe
Intellectual Functioning: IQ between 20-34.
Adaptive Functioning: Individuals require significant support in all areas of daily living.
They may have very limited communication abilities and may rely on caregivers for most
tasks.
Development: Often do not progress to formal academic skills, but can learn basic self-care
and routines with consistent support.
4. Profound
Intellectual Functioning: IQ below 20.
Adaptive Functioning: Individuals require extensive support for all aspects of daily life.
They may have severe limitations in communication and motor skills and typically cannot
live independently.
Development: They may respond to stimuli and engage in simple interactions but require
lifelong care and support.