Hs and Ts of ACLS
Hypovolemia
Loss of fluid volume in the
circulatory system.
Look for obvious blood loss.
Most important intervention is
to obtain IV access and
administer IV fluids.
Use a fluid challenge to
determine if the arrest is
related to hypovolemia
Toxins
Hypoxia
Deprivation of an adequate
oxygen supply can be a
significant contributing
cause of cardiac arrest.
Ensure that the airway is
open.
Ensure adequate ventilation,
and bilateral breath sounds.
Hydrogen Ion
(acidosis)
Hypo/Hyperkalemia
Hypothermia
Obtain an arterial blood gas
to determine respiratory
acidosis.
Both a high and low K+ can cause
cardiac arrest.
If a patient has been
exposed to the cold,
warming measures should
be taken.
Provide adequate
ventilations.
Signs of high K+ include taller,
peaked T-waves, and widening of
the QRS complex.
Use sodium bicarbonate to
prevent metabolic acidosis if
necessary.
Signs of low K+ include flattened
T-waves, prominent U-waves and
possibly widened QRS complex.
Ensure oxygen supply is
connected properly.
Tamponade
Never give undiluted intravenous
potassium.
Tension
Pneumothorax
Accidental overdose : Some of
the most common include:
tricyclics, digoxin,
betablockers, and calcium
channel blockers).
Fluid build-up in the
pericardium results in
ineffective pumping of the
blood which can lead to
pulseless arrest.
Tension pneumothorax
shifts in the intrathroacic
structure and can rapidly
lead to cardiovascular
collapse and death.
Cocaine is the most common
street drug that increases
incidence of pulseless arrest.
ECG symptoms: Narrow QRS
complex and rapid heart
rate.
ECG signs: Narrow QRS
complexes and slow heart
rate.
Physical signs include
bradycardia, pupil symptoms,
and other neurological
changes.
Physical signs: jugular vein
distention (JVD), no pulse or
difficulty palpating a pulse,
and muffled heart sounds.
Physical signs: JVD, tracheal
deviation, unequal breath
sounds, difficulty with
ventilation, and no pulse felt
with CPR.
Poison control can be utilized
to obtain information about
toxins and reversing agents.
Perform: pericardiocentesis
to reverse.
Treatment: Needle
decompression.
Core temp. should be
raised above 86 F and 30 C
as soon as possible.
The patient may not
respond to drug or
electrical therapy while
hypothermic.
Thrombosis
Thrombosis
(heart: acute, massive MI)
(lungs: massive PE)
Causes acute myocardial
infarction.
ECG signs: 12 lead ECG with STsegment changes, T-wave
inversions, and/or Q waves.
Physical signs: elevated cardiac
markers on lab tests, and chest
pain/pressure.
Treatments: use of fibrinolytic
therapy, PCI (percutaneous
coronary intervention).
The most common PCI procedure
is coronary angioplasty with or
without stent placement.
Can rapidly lead to
respiratory collapse and
sudden death.
ECG signs of PE: Narrow
QRS Complex and rapid
heart rate.
Physical signs: No pulse
felt with CPR. distended
neck veins, positive ddimer test, prior positive
test for DVT or PE.
Treatment: surgical
intervention (pulmonary
thrombectomy) and
fibrinolytic therapy.