-SCRIPT EIN Kel.

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1. Pada Tahap pertama adalah perkenalan nama2 anggota kelompok nya

2. Pada tahap yg ini menampilkan 2 presenter untuk membuka presentasi
/OPENING.(YUNI,MARDA)
Assalamualaikum.we are glad to see you all...Let me to introduce my self my name is
marda and Hello my name is Yuni.we would like to present about GASTRITIS.
Before we get down to seriously. Do you know GASTRITIS????
(nanti disini video nya di edit gambar2/2 orang peraga orang sakit perut, stress,
mual,dll)
3. Presenter mengisi suara : (diperagakan lika sedang menunjukkan definisi)
Gastritis is a local inflammation or spreads in the gastric mucosa that develops when
a mucosal protective mechanism is filled with bacteria or irritant substances. (J.
Reves, 1999)
4. Presenter berbicara : Klasification of Gastritis : ACUTE and CRONIC
1. Acute :
1. Drugs such as non-steroidal anti-inflammatory drugs that are irritating the
gastric mucosa
B. Alcoholic beverages
C. Bacterial infections such as H. pylori (most often), E. Colli, Tuberculosis,
etc.
D. Invasive virus by Sitomegalovirus (Giannakis, 2008)
F. Physical stress caused by burns, sepsis, trauma, surgery, respiratory failure,
renal failure, central nervous system damage, and gastrointestinal reflux
(Lewis, 2000)
G. Irritant foods and drinks
H. Bile salts occur in the reflux conditions of bile salts from the small intestine
to the gastric mucosa resulting in a mucosal inflammatory response
(Mukherjee, 2009)
I. Ischemia
J. Direct trauma
2. Cronic :
The exact cause of chronic gastritis gastritis is known, but there are two
important predispositions that may increase the incidence of chronic gastritis,
 ie infection and noninfectious disease (Wehbi, 2008).
2.1 Gastritis infection
A. H. Pylori. Some researchers mention this bacterium is a major cause of
chronic gastritis (Anderson, 2007)
2.2 Gastritis non-infection
A. Immunologic conditions are based on the fact that there are approximately
60% of serum chronic gastritis patients having antibodies to their parietal cells
(Genta, 1996)
B. Chemical gastropathy, associated with chronic bile salt reflux conditions
and contact with NSAIDs or Aspirin (Mukherjee, 2009)
C. Uremic Gastropathy, occurs in chronic renal failure that causes too much
ureum to circulate in the gastric mucosa (Wehbi, 2008)
D. Chronic non-infection granuloma gastritis associated with various diseases
E. Lymphocytic gastritis, often referred to as collagenous gastritis (Sepulveda,
2004)
F. Eosinophilic gastritis (Quentin, 2006)
G. Injury of radiation to the stomach (Sepulveda, 2004)
H. Ischemic gastritis (Sipponen, 1999)
I. Secondary gastritis from drug therapy (Wehbi, 2008)
5. (Latansa dan nde) menunjukkan Pemeriksaan Diagnostik (tapi pengisi suara nya
yuntil):
Acute gastritis -> endoscopic examination followed by histopathological
examination of gastric mucosal biopsy
Gastritis type A -> examination of hydrochloric acid content and examination
of gaston levels.
Gastritis type B -> examination of complete blood cells.
6. So what management or treatment for Gastritis??? (suara marda)
1. Medical (diperagakan lika)
o Antacida
o H2 receptor blocker
o Antibiotics
o Tranguilizer anti-anxiety, antidepressants: if there is a psychic factor.
2. Non Medical (diperagakan depik)
 o Avoid gastritis triggers, such as spicy food, sour, coffee, alcohol, and gas-
containing foods and beverages.
o When vomiting is great, do not eat it first.
o Eat regularly and not excessively. Small portions but often.
o Avoid stress.
o Physical exercise.
7. CLOSING from presenter Oke gais in conclusion we are as nurse or medical team we
must critical think, and very awareness with gastritis disease.
So what should we do? Dijelaskan oleh pengisi suara. Kemudian bilang many thanks
for your attention in our presentation. GOOD BYE. WSAALAM.
Prevention of gastritis (LAILATUR peraga)
1. 1 Eat regularly. Because if eating irregularly or too late to eat and usually wait hungry
first, just eat and eat directly eat too much (Puspadewi, 2009).
2. 2. Avoid smoking. Smoking will damage the protective layer of the stomach. Therefore,
people who smoke are more sensitive to gastritis and ulcers. Smoking will also increase
stomach acid, slows healing and increases the risk of stomach cancer (Yuliarti, 2009).
3. 3. Avoid stress. This is possible because the nervous system in the brain associated with
the stomach, so that if a person experiencing stress, can appear abnormalities in the
stomach. Stress can cause hormonal changes in the body. That change will stimulate the
cells in the stomach which then produce acid in excess. This excessive acid makes the
stomach painful, sore and bloated. This can cause stab wounds in the stomach wall (Sari,
2008).
4. 4. Be careful in taking drugs. The consumption of painkillers, such as nonsteroidal anti-
inflammatory drugs (NSAIDs) such as aspirin, ibuproven (Advil, Motrin etc.), also
naproxen (aleve), which too often can cause gastritis, both acute and chronic gastritis
(Aziz, 2011).
5. 5. Avoid consuming too spicy and sour foods. Drink drinks containing alcohol and
caffeine like coffee. It can increase the production of excessive stomach acid to the end
irritation and decrease the function of the stomach wall (Suratum, 2010)
6. 6. Avoid consuming alcohol, consuming alcohol can irritate (stimulate) and erode the
surface of the stomach (Suratum, 2010).