Sex Addiction: Neuroscience
Etiology and Treatment
Stefanie Carnes, Ph.D., CSAT-S
AAMFT Approved Supervisor
Rates of Sex Addiction Among CD
Patients (Deneke et al. 2014)
Rates of Sex Addiction in Chemical Dependency
Patients
Residential -18 %
Relapse Unit - 19 %
Extended Care Unit – 29 %
“Failure to complete a comprehensive screening for
behavioral addiction may compromise substance use
disorder treatment and maintain a revolving pattern
of substance abstinence and relapse”
Is Sex an Addiction?
DSM III-R contained a category called "non-
paraphilic sexual addiction"
Various authors have argued for different terms
- "Compulsive" (OCD, Coleman, 2003)
- "Addictive" (Fenicehel, 1945, Carnes, 1983)
- "Impulsive" (Barth and Kinder, 1987)
- "Hypersexual" (Stein et al., 2000, Reid/ Kafka)
Criteria across these different conceptualizations are
similar
Is not in DSM-5
ICD-11 draft includes “sexual compulsivity” –
Narrower term “sexual addiction”
American Society of Addiction
Medicine
“Addiction is a primary, chronic disease of brain reward, motivation,
memory and related circuitry. Dysfunction in these circuits leads to
characteristic biological, psychological, social and spiritual
manifestations. This is reflected in an individual pathologically
pursuing reward and/or relief by substance use and other
behaviors.”
“Addiction also affects neurotransmission and interactions between
cortical and hippocampal circuits and brain reward structures, such
that the memory of previous exposures to rewards (such as food,
sex, alcohol and other drugs) leads to a biological and behavioral
response to external cues, in turn triggering craving and/or
engagement in addictive behaviors.”
4 years – 80 neuroscientists
Taken from: http://www.asam.org/advocacy/find-a-policy-statement/view-policy-statement/public-policy-
statements/2011/12/15/the-definition-of-addiction on 9/25/12
DSM-5 Field Study Report for
Hypersexual Disorder
DSM-5 Hypersexual Disorder Field
Trial Report
Reid et al. (2012) conducted a field study to investigate
the “clinical utility, reliability and validity of diagnostic
validity of [hypersexual disorder (HD)] criteria in clinical
settings” for possible inclusion in the DSM-5.
Also explored proposed HD specifiers and their
consequences
Sexual behavior with consenting adults, cybersex, telephone sex
and going to strip clubs
Examined the clinical course of HD
Goal was to examine the inter-rater reliability of
clinicians attempting to diagnose HD.
Reid, R. , Carpenter, B.N., Hook, J.N., Garos, S., Manning, J.C., Gilliand, R., Cooper, E.B., McKittrick, H.,
Davitan, M., & Fong, T. (2012). Report of findings in a DSM-5 Field Trial for Hypersexual Disorder.
Journal of Sexual Medicine, 9, 2868-2877. DOI: 10.111/j.1743-6109.2012-02936.x
DSM-5 Proposed
Criteria for
Hypersexual
Disorder
(Reid et al. (2012)
Method
Included 13 raters from a variety of fields (psychiatry,
psychology, social work, marriage and family therapy, etc)
practicing in outpatient settings
Participants completed the Mini-International
Neuropsychiatric Interview (MINI 6.0) a structured diagnostic
interview at intake to rule out any other psychopathology
that could account for HD symptoms
They also completed the HD Diagnostic Clinical Interview, the
HD Questionnaire (HDQ), the HD Course Questionnaire
(HDCQ), Hypersexual Behavior Inventory (HBI); Sexual
Compulsivity Scale (SCS), NEO Personality Inventory-Revised
(NEO-PI-R), the Hypersexual Behavior Consequences (HBCS)
and the Erotic Preferences Examination Scheme (EPES)
Procedures
Raters were trained on how to complete the
structured diagnostic interviews correctly and to
assess for the proposed HD criteria
One rater completed and scored the initial
interviews of the MINI 6.0 and HD-DCI and another
rater scored it as well
A third rater blind to the initial ratings administered
and scored the HD-DCI two weeks later
Results
Inter-rater reliability: kappa coefficient of .93 among the clinicians
Indicates the diagnostic criteria can be reliably used in patients
Test-Retest Reliability: “high” for the HD criteria after the two week
follow-up (ϕ=.81, p<0.001)
Suggests reliability of the diagnostic criteria over time
Sensitivity=.88, Specificity=.93, Positive Predictive Power=.97,
Negative Predictive Power=.74
Results suggested the proposed HD criteria reflected the presenting
problems well.
Specifiers for HD: Pornography (81.1%) Masturbation (78.3%),
Phone Sex (7.9%), Cybersex (18.1%), Strip Clubs (9.4%), Sex with
Consenting Adults (44.9%)
Diagnostic Validity of HD criteria
High Concurrent Validity-HDQ scores were highly correlated with HBI
(r=.911) and SCS scores (r=.829)
Results Continued
Concurrent Validity
Participants reporting having sex while experiencing negative
emotions had higher Neuroticism scores on the NEO-PI-R.
There was a significant positive correlation between the number
of consequences people reported as a result of their sexual
behaviors and higher levels of hypersexual behaviors.
Clinical Course: 54% of participants reported
“dysregulated sexual fantasies, urges and behaviors prior to
adulthood,” 30% indicated these issues started in their
college years.
82% endorsed a gradual progression of HD symptoms lasting
months to years
48.6% reported a continuous course, while 51.4% reported
episodic symptoms
Conclusions
The researchers suggested the proposed HD
diagnostic criteria could be reliably applied to
people presenting with hypersexual behaviors and
was measuring a valid construct
However, HD was not ultimately included in the
DSM-5.
Why Wasn’t HD Included in the DSM-5?
Reid and Kafka (2014) posited a number of
reasons why Hypersexual Disorder was not
included in the DSM-5
Politics
Previous DSM editors openly criticized the DSM-5
Task Force and Workgroups before its publication
Some members of the Sexual and Gender Identity
Disorders DSM-5 Task Force Committee were
specifically targeted in the media
Some contended the HD diagnosis “confused social
disapproval and morality with issues of health and
disorder” (Wakefield, 2012)
Reid, R.C. & Kafka, M.P. (2014). Controversies about Hypersexual Disorder and the DSM-
5. Current Sexual Health Reports, 6, 259-264. DOI:10.1007/s11930-014-0031-9
Potential Legal Implications & Problems
Concerns about potential misuse in the forensic
community
For example, using an HD diagnosis as mitigating factor
in cases of child molestation
No evidence a pedophilia diagnosis has ever resulted in a
reduced sentence
Authors note a recent field study of HD diagnosis in
sex offenders resulted in very few diagnoses of HD
Criticisms of the Diagnostic Criteria
Belief that the diagnostic criteria did not differentiate
between high sex drives and pathological levels and
activities
Reid and Kafka suggested individual criterion were
“dissected” and rejected while neglecting the fact that
a constellation of at least four of the five symptoms
over 6 months would need to be present for a diagnosis
Some argued hypersexual behaviors could be better
accounted for by another already existing
psychological disorder
Pathologizing Normal behavior?
Some researchers and clinicians argue hypersexual
behaviors are simply variants of normal sexual
behavior that an HD diagnosis is pathologizing
There are also concerns regarding increasing the
number of people diagnosed with a mental illness,
the number of false positives and the number of
people on unnecessary psychotropic medications
Insufficient Empirical Research on HD
Concerns about adding new disorders without
sufficient scientific research
There is a definite lack of epidemiological studies
More studies with objective data (“e.g., genetic
abnormality, deficits in brain function, etc) are
needed as well
Empirical Identification of Psychological Symptom
Subgroups of Sex Addicts: An Application of Latent
Profile Analysis (Nino De Guzman et al. 2015)
There is a sizeable group of sex addicts that probably do not
have other comorbid disorders (Class 1 and 2).
This provides further evidence for the existence of sex addiction
as a discrete disorder, as opposed to merely being
symptomatic of other psychological disorders.
At the same time, about 24% of the sample (Class 4 and Class
5) likely do have other diagnosable conditions (i.e., mood
disorders and anxiety disorders), and thus highlights the
importance of broad-band psychological assessment to
facilitate treatment planning for sex addicts.
Class 1: No comorbid psychopathology (22.7%)
Clinical Profile External Correlates:
Non-significant levels of Some alcohol and drug use
psychopathology symptoms, with Small but positive correlations with negative
average or below average scores on legal consequences
PAI scales Negative correlation with alcohol and drug-
Low levels of distress and related consequences.
psychological disturbance Negative correlation with sex-related
arrests.
Negative correlation with fantasy, pain,
voyeuristic anonymous sex by SDI.
Negative correlations with Core,
Preoccupation, and Affect Disturbance-SAST.
Class 2: Dysfunctional Negative Emotions (14.9%)
Clinical Profile External Correlates:
Mildly elevated cognitive and Positive correlations with drug and
affective symptoms of anxiety chemical use.
Mildly elevated cognitive and Some legal consequences related to drug
affective symptoms of depression and chemical use in general
Mildly elevated levels of traumatic Positive correlation with intrusive-f sex by
stress reaction SDI.
Mild elevation in irritability
Isolation, social withdrawal
Disruptions in thought processes,
difficulties in concentration and
decision-making
Mild elevations in alcohol and
drug use
Class 3: Mild Depression and Substance-Related
Problems (38.1%)
Clinical Profile External Correlates
Mild depression Negative correlations with fantasy-f,
Mildly disturbed concentration and intrusive-f and intrusive-p, and voyeuristic-
decision-making. p sex by SDI.
Mild elevation on drug and
alcohol use problems.
Class 4: Clinical Depression and Anxiety (18.8%)
Clinical Profile External Correlates
Clinically significant cognitive and Some legal implications
affective symptoms of depression Sexual-related convictions.
Clinically significant cognitive and Positive and high correlation with fantasy-
affective symptoms of anxiety f, intrusive-p, voyeuristic-f and voyeuristic-
Clinically significant suicidal p, exhibitionistic –p, trade-p, and
ideation anonymous-p sex by SDI.
Clinically significant symptoms of Positive correlations with core,
traumatic stress preoccupation, loss of control, and
Mildly elevated somatization negative affect sexual behaviors, by
Problems with concentration and SAST.
decision-making
Social withdrawal
Class 5: Severe Psychopathology (5.6%)
Clinical Profile External Correlates:
Highest levels of psychopathology Positive correlations with seductive-f,
Severe levels of anxiety intrusive-f; exhibitionistic-p sex by SDI.
Severe levels of depression Positive and highest sexual preoccupation
Severe levels of traumatic stress and negative relationship disturbance by
reaction symptoms SAST.
Severe levels of suicidal ideation
Clinically significant symptoms of
conversion, somatization, and
health concerns
Clinically significant elevations for
irritability and paranoia
Clinically significant levels of
social detachment and
concentration difficulties
Diagnosis
Differential Diagnosis
DSM-5 - Possibilities:
Other Specified Disruptive, Impulse Control and Conduct Disorder
Other Specified Sexual Dysfunction
Unspecified Paraphilic Disorder
Rule out/ or in:
Antisocial / Narcissistic personality disorder
Paraphilia
Bipolar affective disorder
PTSD
ADHD
Substance induced disorder
Dissociative disorder
Delusional disorder
OCD
Delirium, dementia, or other cognitive disorder or organic condition
Krueger, 2016
“Diagnoses that could refer to compulsive sexual behavior have
been included in the DSM and ICD for years and can now be
diagnosed legitimately in the United States using both DSM-5
and the recently mandated ICD-10 diagnostic coding.
Compulsive sexual behavior disorder is being considered for
ICD-11”
The recommended code, according to the ICD-10-CM index, is
F52.8, which is the code for ‘other sexual dysfunction not due
to substance or known physiological condition’; the inclusion
terms of ‘excessive sexual drive’, ‘nymphomania’ and
‘satyriasis’ are listed under F52.8. DSM-5 also lists ‘other
specified sexual dysfunction’ as F52.8 [13]. This diagnosis may
thus be used for hypersexual disorder.
Krueger, R (2016) Society for the Study of Addiction.
http://onlinelibrary.wiley.com/doi/10.1111/add.13366/full
Differential Diagnosis
Antisocials (especially w/ sex offenders) w/out
addiction
- History of physical abuse
- Lacking remorse an shame
- More force/ violence in the offense
- History of other types of offenses
- More impulsive (not as many sexual urges)
- Distortions and denial
- Decreased amenability for treatment
Differential Diagnosis Continued
Sex Addicts
- High shame
- Emotional and sexual abuse in background
- Highly sexualized (lots of preoccupation)
- Multi-addicted
- Less defenses
- High potential for suicide
- Increased amenability for treatment
Paraphilias are not Sex Addiction
DSM-5 Paraphilias include: exhibitionism,
fetishism, frotteurism, pedophilia, sexual
masochism, sexual sadism, voyeurism, and
transvestic fetishism.
In DSM -5– new definition must include
“psychological distress” or “distress, injury
or death of unwilling persons – or those
not of legal age”
Just because someone has had affairs, used
prostitutes, attended a strip club, uses porn
recreationally…does not mean they are a sex
addict… It is just as important to determine
who is NOT a sex addict as it is to determine
who is.
Etiology
The making of a sex addict
Etiology
Biology/ Neuroscience/ Sexual Conditioning
Family Dynamics/ Attachment
Trauma and abuse
Neuroscience & Sexual Conditioning
Brain Regions Involved in
Addiction
Mesolimbic Dopamine (DA) Pathway:
Connects the ventral tegmental area to the nucleus accumbens (NAc)
“Reward Center” tied to pleasure, reinforcement learning & impulsivity
Amygdala:
Positive & negative emotional memory
Hippocampus:
Processing & retrieval of long-term memories
Prefrontal Cortex:
Coordinates & determines judgment & behavior
Evolutionary Function of Reward
System
Mesolimbic Dopamine Pathway is activated by salient
survival-based stimuli (sex, food, nurturing, etc.).
This system evolved to reward and encourage the
organism to seek out activities necessary for survival.
Addiction in the brain
Evolutionary Adaptive System Hijacked
Addiction is a Brain Disease –
Volkow et al. 2016
Three re-occurring phases (called the “addiction
circle”):
1 – Binge and Intoxication
2 – Withdrawal and Negative Affect
3 - Anticipation and Craving
Binge and Intoxication
Dopamine released in reward system (nucleaus accumbens )
Repeated exposure to rewards leads to cue responsivity over
time – which predicts increased intake of the substance/
behavior (classical conditioning)
Leads to “Incentive Sensitization” – Robinson and Berridge
(1993) – stimuli associated with the reward become “cues”
that trigger enhanced dopamine release signaling incentive
salience and induced “wanting” (clinically described as
craving).
This causes neural and molecular changes in reward system
(neuroplasticity) in many brain regions
Results in the “down-regulation” of dopamine resulting in
tolerance
Withdrawal and Negative Affect
Withdrawal symptoms and negative affect are
consequences of the brains natural compensatory
response to excessive dopamine
Brain is trying to maintain homeostasis
Natural rewards are experienced as less rewarding
by addicted subjects than healthy subjects
Motivates further reward seeking behavior to stop
negative affect (negative reinforcement)
Preoccupation and Anticipation Phase
Preoccupation with obtaining the reward
Craving
Changes in pre-frontal regulatory circuits that leads
to impaired response inhibition
How does Dopamine Down
Regulation Work?
Learning, Memory & Motivation “Wanting”
Dopamine interacts with glutamate to produce
a hyper-excitable state that enhances the
responsiveness of the mesolimbic dopamine
reward system.
Hippocampus records memories of intense reward.
Amygdala records memories of environmental cues associated
with the intense reward.
Brain mistakenly treats the highly rewarding substance or
behavior as necessary for survival, reducing “top down”
inhibitory control, increasing impulsivity and motivating further
action to seek out the source of pleasure.
Tolerance Reduces “Liking”
Repeated use over time leads to over-stimulation
of the dopamine reward system.
To maintain homeostatic balance, the brain eventually down-
regulates dopamine receptor availability in the striatum, producing
an altered set point for pleasure.
Substance or behavior no longer produces the intense pleasure that
it did originally (tolerance).
Other "normal" sources of pleasure don't produce a noticeable
impact on the down-regulated reward system, leaving the individual
feeling anxious, depressed, dysphoric & irritable (withdrawal).
Dopamine Down-Regulation is a Well-Established Finding in
Addiction Research
Nucleus Accumbens—Brain’s Reward Center
Red
indicates
high number
of receptors Normal Obese
for
dopamine
Hans Breiter, director of the
Motivation and Emotion
Neuroscience Center at
Massachusetts General Hospital
Alcoholic Cocaine
People short of dopamine have difficulty
feeling joy.
“An orgasm is the primary natural blast of
dopamine available to all of us. Accordingly, J.R.
Georgiadis (2006) scanned the brains of people
having orgasm. He said they resembled scans of
heroin rushes. These individuals experienced one of
the most addictive substance ever produced:
dopamine.” (p.137).”
Blum, K., Chen, A., Giordano, J., Borsten, J., Chen, T., Hauser, M., Simpatico, T., Femino, J., Braverman, E. R., &
Barh, D. (2012). The addictive brain: All roads lead to dopamine. Journal of Psychoactive Drugs, 44 (2), 134–
143. DOI: 10.1080/02791072.2012.685407
Effects of Drugs on Dopamine Levels
1100 Accumbens AMPHETAMINE Accumbens
COCAINE
1000 400
% of Basal Release
900
% of Basal Release
800 DA
DA 300 DOPAC
700 DOPAC HVA
600 HVA
500 200
400
300
200 100
100
0
0 1 2 3 4 5 hr 0
0 1 2 3 4 5 hr
Time After Amphetamine Time After Cocaine
250 250
NICOTINE Accumbens ETHANOL
% of Basal Release
200 Accumbens Dose (g/kg ip)
200
% of Basal Release
Caudate
0.25
150 0.5
1
150 2.5
100
100
0
0 1 2 3 hr 0
0 1 2 3 4hr
Time After Nicotine Time After Ethanol
Source: Di Chiara and Imperato
Natural Rewards Elevate Dopamine Levels
FOOD SEX
200 200
DA Concentration (% Baseline)
NAc shell
150 150
% of Basal DA Output
100 100
Copulation Frequency
15
Empty 10
50
Box Feeding
5
0 0
0 60 120 180 Scr Scr Scr Scr
BasFemale 1 Present Female 2 Present
Time (min) Sample 1 2 3 4 5 6 7 8 9 10 11 12 13 14 15 16 17
Number
Mounts
Intromissions
Ejaculations
Source: Di Chiara et al. Source: Fiorino and Phillips
Koob: “The dark side of addiction”
When the reward center can no longer be returned to
it’s homeostatic set point it enters an “allostatic” state
Reward system has an altered set point
Leaves the individual susceptible to dependence and
relapse
Withdrawal is not about the physiological effects of
a specific substance – it is the negative affect
resulting from this allostatic state
Dr. David Linden – Professor of Neuroscience, John Hopkins
School of Medicine
"The truth is that just liking sex a lot doesn't make you a
sex addict, and just cheating or engaging with
prostitutes or other anti-social behavior doesn't make
you a sex addict. If you are a sex addict, just like a
heroin addict ... you are at the point where you are
having sex not because you are deriving pleasure from
it, but because you need to do that just to fall asleep at
night and face the day, and not have withdrawal
symptoms. So while true sex addiction is rare, it is one
of many very real addictions that stem from the way
the human brain feels - or doesn't feel - pleasure.“
- The Compass of Pleasure: How Our Brains Make Fatty Foods, Orgasm, Exercise,
Marijuana, Generosity, Vodka, Learning, and Gambling Feel So Good
Kuhn – High Porn Consumption Associated
w/Grey Matter Volume Reduction
2014
Brain Structure and Functional Connectivity Associated With
Pornography Consumption: The Brain on Porn (2014)
Higher hours per week/more years of porn viewing
correlated with a reduction in grey matter in sections
of the reward circuitry (translates into sluggish reward
activity, or a numbed pleasure response –
desensitization)
Simone Kühn - "That could mean that regular
consumption of pornography more or less wears out
your reward system.“
Simone Kühn continued - "We assume that subjects with
a high porn consumption need increasing stimulation to
receive the same amount of reward.”
Voon – Neural Mechanisms Underlying CSB
Similar to Those in Found in CD
2014
Neural Correlates of Sexual Cue Reactivity in Individuals
with and Without Compulsive Sexual Behaviors (2014 –
Voon et al. Cambridge University)
Compulsive porn users react to porn cues in the
same way that drug addicts react to drug cues
Compulsive porn users craved porn (greater
wanting), but did not have higher sexual desire
(liking) than controls. This finding aligns perfectly
with the current model of addiction.
Over 50% of subjects (average age: 25) had
difficulty achieving erections with real partners,
yet could achieve erections with porn
Enhanced Attentional Bias towards Sexually Explicit Cues in
Individuals with and without Compulsive Sexual Behaviors –
Voon et al. 2014
“Our findings of enhanced attentional bias in CSB
subjects suggest possible overlaps with enhanced
attentional bias observed in studies of drug cues
in disorders of addictions. These findings converge
with recent findings of neural reactivity to sexually
explicit cues in CSB in a network similar to that
implicated in drug-cue-reactivity studies and
provide support for incentive motivation theories
of addiction underlying the aberrant response to
sexual cues in CSB.”
Gola et al. (2017)
Gave fMRIs to 28 men in treatment for problematic pornography use (PPU)
and 28 men without PPU to examine ventral striatal responses to “erotic
and monetary stimuli”
Wanted to differentiate “cue-related ‘wanting’ from reward-related
‘liking’”
Participants completed an incentive delay task during the fMRI and were
given “erotic or monetary rewards preceded by predictive cues”
PPU group had higher activation in the ventral striatum for cues that
predicted erotic stimuli but not for cues that predicted monetary reward or
to the actual erotic pictures
Authors argued this is “consistent with the incentive salience theory of
addiction”
Sensitivity to erotic stimuli cues was related to increased motivation to see
the erotic stimuli (suggests “higher wanting”), higher pornography use,
severity level of PPU and more frequent masturbation
Findings congruent with research on gambling and substance addictions
suggesting PPU may be a behavioral addiction
Gola, M., Wordecha, M., Sescousse, G., Lew-Starowicz, M., Kossowski, B., Wypych, M., ... & Marchewka, A. (2017). Can
pornography be addictive? An fMRI study of men seeking treatment for problematic pornography use. bioRxiv, 057083.
Ji-Woo Seok and Jin-Hun Sohn of the Brain Research
Institute at Chungnam National University in South Korea
Sex addicts focus a higher-than-normal share of their
attention on addiction related cues (i.e., pornography),
doing so in the same basic ways and to the same basic
degree as other addicts.
The brain response of sex addicts exposed to sexual stimuli
(i.e., pornography) mirrors the brain response of drug
addicts when exposed to drug-related stimuli. For example,
the dorsal orbital prefrontal cortex lights up just as it does
with substance addicts. Equally important is the fact that this
region goes below baseline for neutral stimuli, the same as
with substance abusers. In other words, the dorsal orbital
prefrontal cortex overreacts to addiction cues and
underreacts to neutral cues in all forms of addiction,
including sexual addiction.
Banca et al. (2016)
Examined whether men with CSB showed more of a preference for “sexual novelty and stimuli
conditioned sexual rewards” compared to a healthy control group
CSB group:
Had a stronger preference for novel sexual images in comparison to control images
Demonstrated a preference for cues that had been conditioned to sexual and monetary
rewards over neutral outcomes
This result was not observed in the control group
Had higher levels of dorsal cingulate habituation during an fMRI when presented with
repeated sexual images compared to monetary images
Level of habituation to sexual images was positively correlated with self-reported
preference for sexual novelty
Had an early attentional bias to sexual cues compared to control group that significantly
correlated with higher levels of approach behaviors towards cues conditioned to sexual images
Authors concluded the CSB participants had a “dysfunctional enhanced preference for sexual
novelty possibly mediated by greater cingulate habituation” as well as an overall enhanced
reaction to rewards
The novelty seeking and cue conditioning found in CSB participants is similar to results seen in
studies on substance addictions
Banca, P., Morris, L. S., Mitchell, S., Harrison, N. A., Potenza, M. N., & Voon, V. (2016). Novelty, conditioning
and attentional bias to sexual rewards. Journal of psychiatric research, 72, 91-101.
Hypofrontality
Cognitive problems such as forgetting responsibilities
Problems with working memory
Increased impulsivity
Decreased cognitive flexibility
Decreased ability to multi-task
Difficulties with sustaining attention
Slower learning
Difficulties making decisions
Decreased emotional regulation
Our clients experience
Powerful sexual conditioning and learning
Neuroplastic change
Structural changes in the brain
Deficits in areas of functioning (e.g. memory, decision
making)
Over 30 articles on the neuroscience of sex addiction…
Embedded in a large body of research on behavioral
addictions (130 behavioral addiction articles - e.g. 70 brain
articles on internet addiction)
Longitudinal research in other areas
Porn and the Brain
Novelty and the “Coolidge” Effect
Gary Wilson
Presentation of a new potential mate creates a surge of
dopamine in the brain
Becomes harder to mate with the same old partner (less
dopamine is released)
Gary Wilson “Endless online mates keep dopamine
surging”
“Males need time to recover their potency and vigor
after overriding their sexual satiation mechanisms with
dopamine/novelty.”
“Porn Induced” Erectile Dysfunction – due to
desensitized dopamine system in the brain – as
opposed to blood flow in penis as in natural later onset
Hilton – Pornography Potentially
Addictive
2013
Supernormal Stimulus
A supernormal stimulus or superstimulus is an
exaggerated version of a stimulus to which there is an
existing response tendency, or any stimulus that elicits a
response more strongly than the stimulus for which it
evolved.
Nickolaas Tinbergen discovered
animals (birds, gypsy moths etc)
could be fooled into preferring fake
mates and eggs.
It’s not how much time is spent that
leads to problematic use….
Important findings in this study are that neither time spent viewing porn on the
Internet nor personality factors were associated the level of reported
problems with Internet porn use
Instead, it was intensity of the experience and amount of novelty
(different applications opened)
“It has generally been assumed that predisposing personality problems are
what make porn addiction possible, but it may be dopamine levels, quite
apart from personality.”
As it turns out, the level of reported psychological problems (e.g., social
anxiety, depression, and compulsivity) appears to be related to how
intense the arousal produced, and the number of applications used
(degree of novelty).
"Although we did not examine brain correlates of watching Internet
pornographic pictures in our study, we found the first experimental evidence
for the potential link between subjective reactivity on Internet pornographic
stimuli and a tendency toward cybersex addiction.“
Brand, M., Laier, C., Pawlikowski, M., Schächtle, U., Schöler, T., & Altstötter-Gleich, C. (2011). Watching pornographic pictures on
the internet: Role of sexual arousal ratings and psychological–psychiatric symptoms for using internet sex sites
excessively. Cyberpsychology, Behavior, and Social Networking, 14(6), 371-377.
Sexual Conditioning
“Erections may become conditioned to aspects of
VSS [porn] that do not transition easily to real-life
partner situations. Sexual arousal may be conditioned to
novel stimuli, including particular sexual images, specific
sexual films or even non-sexual images. It is conceivable
that experiencing the majority of sexual arousal within
the context of VSS may result in a diminished erectile
response during partnered sexual interactions. Similarly,
young men who view VSS expect that partnered sex will
occur with themes similar to what they view in VSS.
Accordingly, when high stimulation expectations are not
met, partnered sexual stimulation may not produce an
erection.”
Prause & Pfaus, 2015 Veiwing sexual stimuli associated with greater
sexual responsiveness not erectile dysfunction
Contemporary Vs. Classic SA
Reimersma & Sytsma (2013)
Classic:
History of abuse
Insecure attachment
Poor impulse control
Cross Addictions
Co-morbid mood disorders
Used to soothe toxic emotions
Contemporary
Rapid onset
Due to explosive growth of internet technology
Chronic exposure to graphic content online
Content – unique, intense, graphic, limitless novelty
Culture – trending towards virtual and non-relational sex
Early exposure to graphic sexual material
Sexual conditioning
Less trauma history/ attachment problems
May not be having sex (or may never have had sex)
May not be able to perform – can include performance
anxiety, unrealistic performance standards
Giordana and Cashwell Prevalence
study 2017
339 college students surveyed and found that 10.3% scored
in the clinical range for cybersex addiction. Further, we
found significant gender differences among the clinical and
non-clinical range groups as males were more likely to score
in the clinical range for cybersex addiction.
From Table 1 in the full paper (which is available in the
SASH journal Sexual Addiction & Compulsivity)
Percentage of men in the clinical range - 19%
Percentage of women in the clinical range - 4%
Psychological Correlates of Internet
Porn Use
Levin, Lillis and Hayes (2012) found the following
correlates of increased porn use in college males:
Depression
Anxiety
Stress
Poor social functioning
Lam, C.B. & Chan, D.K.S. (2007). The use of cyberpornography by young men in Hong Kong: Some psychosocial correlates.
Archives of Sexual Behavior, 36, 588-598
Levin, M.E., Lillis, J. & Hayes, S.C. (2012). When is online pornography viewing problematic in college males? Examaning the
moderating role of experiential avoidance. Sexual Addiction & Compulsivity, 19, 168-180
Porn Use & Erectile Dysfunction in
Young Men
Foresta and colleagues (2011) studied 28,000 Italian
men and found higher levels of porn use was associated
with higher levels of erectile dysfuction in young men
Landripet and Stulhofer (2015) found that moderate
(but not high) levels of porn consumption were related
to higher chances of young Croatian men having
erectile difficulties
Voon (2014) found that over half (11 of 19) men with
compulsive porn use reported erectile dysfunction
First time in history – widespread youthful ED in young
men
Rebooting for PIED
General recovery after 2 months of no porn or
masturbation
Older guys are recovering faster than those that
wired their brains to internet porn during
adolescence
Fapstronauts / “No Fap” community on Reddit
Rebootnation.org
Yourbrainonporn.com
Families & Attachment
Circumplex
Copyright D.H. Olson
Measure Flexibility
Copyright D.H. Olson
Measure Cohesion
Copyright D.H. Olson
Families of Sex Addicts
77%
RIGID
87%
DISENGAGED
Copyright D.H. Olson
Significant differences in Attachment
Styles of Sex Addicts
Attachment varies by gender and sexual
orientation
Anxious
Avoidant
Correlates of Porn Use in Young Men
2014
Szymanski and Stewart-Richardson (2014)
Higher frequencies of pornography use and
problematic porn use were related to:
Avoidant attachment style
Anxious attachment style
Decreased sexual satisfaction
Increased relational problems
Szymanski, D.M. & Stewart-Richardson, D.N. (2014). Psychological,
relational, and sexual correlates of pornography use on young adult
heterosexual men in romantic relationships. The Journal of Men’s
Studies, 22(1), 64-82
Trauma
Trauma and Abuse History
Most came from families were abuse and trauma
were present.
72% experienced physical abuse
81% experienced sexual abuse
97% experienced emotional abuse
In addition, they came from families where shame
was present.
“CSB (Compulsive Sexual Behavior) has been
strongly linked to early childhood trauma or abuse,
highly restricted environments regarding sexuality,
dysfunctional attitudes about sex and intimacy, low
self-esteem, anxiety, and depression.”
Coleman, E. (1992). Is your patient suffering from compulsive sexual behavior?
Psychiatric Annals , 22(6), 320-325.
“Sexual addiction is strongly anchored in shame
and trauma. Research conducted over the last
fifteen years has consistently shown the prevalence
of emotional, physical, and sexual abuse in this
population.”
Cox, R. P., & Howard, M. D. (2007). Utilization of EMDR in the treatment of sexual
addiction: A case study. Sexual Addiction & Compulsivity, 14(1), 1-20. doi:
10.1080/10720160601011299
Recent Research
Recent Study (2012) found 39% of gay and bisexual
men with compulsive sexual behavior had experienced
childhood sexual abuse
These findings are “largely consistent with previously
studied self-identified community samples of individuals
with CSB (Black et al., 1997; Kafka & Prentky, 1992)
This finding is in line with Briere and Runtz’s (1990)
report that childhood sexual abuse was uniquely
associated with maladaptive sexual behavior, and with
previous literature supporting childhood sexual abuse
as a possible etiological factor in CSB development
(Perera et al., 2009) (p.419).”
Blain, L. M., Muench, F., Morgenstern, J., & Parsons, J. T. (2012). Exploring the role of child sexual abuse and
posttraumatic stress disorder symptoms in gay and bisexual men reporting compulsive sexual behavior. Child
Abuse & Neglect, 36(5), 413-422.
Assessment
Assessment Tools
SAST-R 2.0
PATHOS
SDI-R
PATHOS
1) Do you often find yourself preoccupied with sexual thoughts?
(Preoccupied)
2) Do you hide some of your sexual behavior from others? (Ashamed)
3) Have you ever sought help for sexual behavior you did not like?
(Treatment)
4) Has anyone been hurt emotionally because of your sexual
behavior? (Hurt)
5) Do you feel controlled by you sexual desire? (Out of control)
6) When you have sex, do you feel depressed afterwards? (Sad)
SDI
Comprehensive battery of tests
SAST
Diagnostic Criteria, Anorexia, Collateral Indicators
Co-morbid Addiction screen
Financial Costs
Consistency, exaggeration
Attachment Style
Readiness for Change
MULTIPLE ADDICTIONS
Binge / Purge Cycle
Acting Out
Extreme Control Out of Control
Dieting Eating
Sex Avoidance Sex & Romance
Saving / Hoarding Alcohol
Drugs
Risk Aversion
Spending / Debting
Compulsive Athleticism Risk-taking
Work
Gambling
Acting In
Treatment
When Substance Addiction is Present…
Chronology of treatment is vital…
Client must be carefully detoxed and stabilized
Clients may be initially screened and assessed to see if
sex addiction is present
Therapeutic alliance and supportive community
established
After client is stable and is not at risk of elopement and
has increased capacity for emotional regulation
Proceed slowly on sexual issues
Sexual issues may be associated with trauma and shame
Manage triggers, cues
Provide support
Treatment
Programmatic care
Group
12 step support
Educational component
Celibacy agreement
Sexual health plan
Task methodology
12 step
Mindfulness, CBT
IFS, Trauma treatment, EMDR, SE
Family / Couple treatment
Partner Trauma Treatment
Disclosure
Task Methodology: The Process
For Healing…Three Legged Stool
Addict’s therapist
Individual therapy
Support Groups/ 12 step support
Partner’s therapist
Individual therapy
Support Groups
Couples therapist
Question and Answer
Thank you!