Definition
 Rhythmic, cyclic movement of the limbs in
  relation to the trunk resulting in forward
  propulsion of the body.
 Gait is your manner, pattern, or style of
  walking. An easy walking gait is normal and
  healthy, but injury, illness, or muscle weakness
  can cause pain or functional mobility loss that
  affects your gait
NORMAL GAIT REQUIRES
 Normal functioning of musculoskeletal system of
 lower limbs & spine.
 Good sensory feedback from propioceptive sensation
 from feet and the joints.
 Visual ,labrinthine sensory inputs & co ordination
 add smoothness, rhythm & elegance to the human
 gait.
GAIT TERMINOLOGY
➢ Base of support
➢ Step length
➢ Stride length
➢ Gait cycle
➢ Cadence
➢ Walking velocity
➢ Double limb support
➢ Single limb support
➢ Ground reaction force vector
BASIC GAIT TERMS
Base of suppport:
 Distance between a person’s
  feet while standing or during
  ambulation.
 Provides balance & stability to
  maintain erect posture.
 Normally 2-4 inches from
  heel to heel.
Step length
 Linear distance along the line
 of progression of one foot
 travelled during one gait
 cycle.
 Approximately 15 inches.
Stride length
 Linear distance in the plane of
  progression between
  successive point of foot to
  floor contact of the same foot.
 Normally 27 – 32 inches.
Gait cycle
 Period of time from one heel strike to next heel strike of
  the same limb
Cadence
 It is measured as the number of steps / sec or per
  minute.
 Approximately 70 steps per minute.
 It may variable for person to person.
Double limb support
 During normal gait, for a moment , two lower extremities are
 in simultaneous contact with the ground.
 During this period, both legs support the body weight.
 Happens between push off & toe off on same side and heel
 strike & foot flat on the contra lateral side.
GAIT CYCLE COMPONENTS
1. Stance phase :60% of the gait cycle.
2. Swing phase :40% of the gait cycle.
STANCE PHASE
➢Heel strike
➢Foot flat
➢Midstance
➢Heel off
➢Toe off
Swing PHASE
➢ Acceleration
➢ Midswing
➢ Deceleration
Heel strike phase:
 Beginning of stance phase when
 the heel contacts the ground.
 Begins with initial contact & ends
 with foot flat
Foot flat:
  It occurs immediately following heel
   strike
  It is the point at which the foot fully
   contacts the floor.
Mid stance:
 It is the point at which the body passes
  directly over the supporting extremity.
                                             16
Heel off:
 The point following midstance the heel of the
  reference extremity leaves the ground.
                                                  17
Toe off
 The point following heel off when only the
 toe of the reference extremity is in contact
 with the ground.
HIP : STANCE PHASE
PHASE                 NORMAL MOVEMEMT             NORMAL MUSCLE ACTION
Heel strike to foot   30* flexion                 Erector spinae,gluteus
flat                                              maximus,hamstrings.
Foot flat to          30*flexion-(neutral)        Gluteus maximus at beginning to
midstance                                         oppose flexion movement, then
                                                  activity ceases as moment changes
                                                  from flexion to extension
Midstance to heel     extension                   No activity
off
Heel off to toe off   10* hyperextension to neutral Iliopsoas,adductor
                                                    magnus,adductor longus
    KNEE : STANCE PHASE
PHASE                      NORMAL                  NORMAL MUSCLE ACTION
                           MOVEMEMT
Heel strike to foot flat   0*-15* flexion          Quadriceps contracts initially to hold
                                                   knee in extension & then eccentrically
                                                   oppose the flexion movement to
                                                   controll amount of flexion.
Foot flat to midstance      15*flexion-            Quadriceps contract in early part,&
                           5*extension             then no activity is required
Midstance to heel off      5* of flexion-neutral   No activity required
Heel off to toe off        0*-40* flexion          Quadriceps required to control amount
                                                   of knee flexion
ANKLE & FOOT : STANCE PHASE
 PHASE                     NORMAL MOVEMENT              NORMAL MUSCLE ACTIVITY
 Heel srike to foot flat   0*-15* plantar flexion       Eccentric action of tibialis
                                                        anterior oppose plantar flexion
                                                        movement
 Foot flat to midstance    15*plantar flexio-10*dorsi   Gastronemius & soleus act
                           flexion                      eccentrically to oppose
                                                        dorsiflexion movement &
                                                        control tibial advance
 Midstance to heel off     10*-15* dorsiflexion              same as above
 Heel off to toe off       15*dorsiflexion to 20*       Gastronemius,soleus,peroneus
                           plantar flexion              brevis & longus,flexor hallusis
                                                        longus contract to plantar flex
                                                        the foot
Swing phase
Acceleration phase:
• It begins once the toe leaves the ground &
  continues until mid-swing, or the point at
  which the swinging extremity is directly
  under the body.
Swing phase
Mid-swing:
   It occurs approximately when the reference
    extremity passes directly under the body.
   It extends from end of acceleration to the
    beginning of deceleration
                                                 23
 Swing phase
Deceleration
 It occurs after mid-swing
 when the referance extremity is
 decelerating in preparation for heel
 strike.
                                        24
   HIP : SWING PHASE
PHASE                 NORMAL                NORMAL MUSCLE ACTION
                      MOVEMENT
Acceleration to mid   20*-30* flexion       Hip flexor activity to initiate swing
swing                                       iliopsoas,rectus
                                            femoris,gracilis,sartorius,tensor fascia
                                            lata
Midswing to           30*flexion –neutral   hamstrings
deceleration
 KNEE :SWING PHASE
PHASE             NORMAL MOVEMENT            NORMAL MUSCLE ACTION
Acceleration to   40*-60* flexion            Little activity in quadriceps,biceps
mid swing                                    femoris(short head),gracilis,
                                             sartorius contract concentrically
Midswing          60*flexion-30* extension
Deceleration      30*-0* extension           Quadriceps contract concentrically
                                             to stabilize knee in extension in
                                             preparation for heel strike
Ankle & foot : swing phase
PHASE             NORMAL MOTION             NORMAL MUSCLE ACTION
Acceleration to   Dorsiflexion to neutral   Dorsiflexors contract to bring the
midswing                                    ankle in neutral & prevent toes
                                            from dragging on the floor
Mid swing to      Neutral                   dorsiflexion
deceleration
DIFFERENCE BETWEEN
WALKING AND RUNNING
Walking : Always a double support phase
          no flight phase
Running
No double support phase, always flight phase
                      BODY
  PASSENGER UNIT                LOCOMOTOR
                                   UNIT
       HEAD                       PELVIS
       ARM                        LOWER
      TRUNK                        LIMBS
                                FUNCTIONING
   HAS NO ROLE .                   SYSTEM
GOES ALONG THE RIDE
                       STANCE               SWING
                        PHASE               PHASE
DETERMINANTS OF GAIT
I. Displacement of center of gravity (COG).
II. Factors responsible for minimizing
  displacement of center of gravity.
CENTER OF GRAVITY
 It is an imaginary point at which all the weight of the
  body is concentrated at a given instant.
 Center of gravity lies 2 inches in front of the second
  sacral vertebra.
 Centre of gravity follows vertical displacement and
  horizontal displacement
O V E R A L L D IS P L A C E M E N T
   Sum of vertical & horizontal
    displacement                                  Horizontal
                                                    plane
   Figure ‘8’ movement of Center of
    Gravity as seen from Antero
    Posterior
  These displacement require energy
   “Greater the displacement more      Vertical
                                        plane
                  energy is needed”.
Factors responsible for minimizing
the displacement of centre of gravity
 Major determinants:
       Pelvic Rotation (transverse plane)
       Pelvic Lateral Tilt (Obliquity)
       Knee Flexion During Stance
       Ankle Mechanism (Dorsiflexion)
       Ankle Mechanism (Plantarflexion)
       Step Width
 Minor determinants:
  1. Neck movement.
  2. Swinging of arms.
  1. Pelvic rotation
Rotation of pelvis in Horizontal plane in swing phase, total of
8 degree
         ➢Decrease angle of hip flexion & extension
         ➢Enables a longer step length without further
         lowering of Center of gravity
2. Pelvic tilt
•The pelvis slopes downwards laterally towards the leg which
is in swing phase
•Reduces the vertical movements of the upper body, and
thereby increases energy efficiency.
•Decrease the displacement of Center of gravity
 3. Knee flexion in stance
•As the hip joint passes over the foot during the support phase, there
is some flexion of the knee. This reduces vertical movements at the
hip.
•Decrease the displacement of Centre of Gravity
4. Ankle mechanism
•Lengthen the leg at heel strike
•Reduce the lowering of Centre of gravity, hence smoothen the
curve of Center of gravity.
5. Foot mechanism
•Lengthen the leg at toe off as ankle moves from dorsiflesion
to planter flexion
•Reduce the lowering of Centre of Gravity, hence smoothen
the curve of Centre of Gravity
Trunk and Arms
 • The trunk, arms and shoulders also rotate to ensure
   balance
 • Upper limb swings opposite to stance leg to produce a
   smooth balanced gait.
6. Lateral displacement of body
•In normal gait, width of walking base is narrow,
decrease the lateral displacement of Centre of Gravity
•Decrease muscular energy consumption due to decrease
lateral acceleration & deceleration
 Due to complex interaction of muscular activity & joints
 motion in lower limb Centre of Gravity follows a smooth
 sinusoidal curve.
 It reduce the significant energy consumption of
 ambulation.
GAIT IN CHILDREN (<2years)
 Gait of small children differs from that of adult
 The walking base is wider.
 The stride length & speed are lower & the cycle time
  shorter(higher cadence).
 Small children have no heel strike, initial contact being made
  by flat foot.
 There is very little stance phase and knee flexion.
 The whole leg is externally rotated during the swing phase.
 There is an absence of reciprocal arm swinging.
GAIT IN ELDERLY
 The age related changes in gait takes place in decade
 after m 70yrs.
 There is a decreased stride length, increased cycle
 time(decreased cadence).
 Relative increase in duration of stance phase of gait
 cycle.
 The speed almost always reduced in elderly people.
 Reduction in total range of hip flexion & extension,
 Reduction in swing phase and knee flexion
Function of the 6 determinants of gait:
1) Increase the efficiency and smoothness of gait.
2) Decrease the vertical and lateral displacement of
    center of gravity.
3) Decrease the energy expenditure.
4) Make gait more graceful.
                   GAIT ANALYSIS
 KINEMATIC                         KINETIC GAIT
GAIT ANALYSIS                        ANALYSIS
QUALITATIVE     QUANTITATIVE
KINEMATIC GAIT
                                  KINETIC GAIT ANALYSIS
ANALYSIS
 Describe the movement            Determine the force that
 pattern without regard for the    are involved in the gait.
 force involved in producing
 the movement
Gait analysis
 Observational method- naked eye examination
 Photographic method- television , video, movie analysis
 Force plate study method-ground reaction force method
 Electromyographic study (EMG)
 Electrogoniometric study
 Energy expenditure/requirement method
 Multichannel funtional electrical stimulation
 method(MFES)
Clinical gait analysis
1. Observational gait data:(Qualitative)
   Clinician watches patients walk
Advantage:
1. Require little or no instrumentation
2. Inexpensive
3. Yield general description of gait variables
    Clinical gait analysis
2. Gait parameters (Quantitative)
•    The gait parameter measurement are made by
     timing progress over a 16m walkway & identifying
     events by means of foot switch system.
•    These instrument identify the part contacting the
     ground with data transmitted by telemetry.
•Photographic methods are most
accurate.
•After film development, each frame
is analysed using vanguard motion
analyser and sonic digitizer.
4. Force plate data
   It represent the ground reaction force of walking
    generated by force plate
•   Therapiest observe the patient and walking
    pattern.
Electrogoniometer
                     It is used to study
                      the joints during
                      gait.
5. Energetics
 Deals with measurement of oxygen consumption
  during a specific task
 Oxygen uptake is inversely related to the efficiency of
  gait.
PATHOLOGICAL GAIT
➢ Scissoring gait      ➢ Knock knee gait
➢ In toeing gait       ➢ Genu recurvatum gait
➢ Out toeing gait      ➢ Short limb gait
➢ High stepping gait   ➢ Quadricep gait
➢ Circumduction gait   ➢ Gluteal medius gait
➢ Waddling gait        ➢ Gluteal maximus gait
➢ Trendelenberg gait   ➢ Stiff hip gait
➢ Drunkers gait
➢ Festinant gait
➢ Antalgic gait
ANTALGIC GAIT
 Gait pattern in which stance phase on affected side is
 shortened due to pain in the weight bearing limb.
 There is corresponding increase in stance phase on
 unaffected side
 Common causes: Osteoarthritis, Fractures, tendinitis,
 Inflammation in affected limb.
TRENDELENBERG GAIT
 Any condition which distrupts the osseo-muscular
 mechanism between pelvis and femur
 Weak abductors (power),acetabulo femoral articulation
 defect(fulcrum),defective lever system causes trendelenberg
 gait.
 Here the abductor action in pulling the pelvis downwards in
 stance phase becomes ineffective and the pelvis drops on the
 opposite side causing instability.
 To prevent this body lurches on the same side.
Trendelenberg gait
 Usually unilateral
 If bilateral = waddling gait
 Causes :
1. Weak abductors :poliomyelitis . muscular dystrophies,
   motor neuron disease
2. Defective fulcrum: Congenital dislocaion of hip(CDH),
   pathological dislocation of hip
3. Defective lever : Fracture neck of femur, Perthes disease,
   Coxa vara.
Circumduction gait
 In hemiplegic patients
 To avoid the foot from
 scrapping the ground, the hip
 and the lower limb rotates
 outward.
High stepping gait
 Due to foot drop
 On attempt of heel strike, the toe drops to the ground
  first.
 To avoid this the patient
  flexes the hip and knee
  extensively to raise the
  foot and slaps it on the
  floor forcibly.
Scissoring gait
 Here one leg crosses directly over the other with each
  step due to adductor tightness.
 Seen in Cerebral palsy
Drunkers or reeling gait
 Patient tends to walk irregularly on wide base, swinging
  sideways without stability and balance.
 Caused due to cerebellar lesion.
 With unilateral lesion of cerebellum, balance is lost towards the
  side of the lesion.
Genu recurvatum gait
  In Paralysis of hamstring muscles the knee goes in for
   hyper extension while transmitting the weight in mid
   stance phase.
  Seen in poliomyelitis
Short limb gait
 Shortening less than 1.5 cm compensated by pelvic tilt, and
 shortening upto 5 cm compensated by equinus.
 Shortening more than 5 cm the patient dips his body on that
 side.
Festinant gait
 Seen in Parkinson's disease
 Steps are short that the feet barely clears the ground.
Quadriceps gait
 Normally the knee is locked by the quadriceps contraction
  while transmitting weight to the lower limb during
  midstance.
 Hence patient with weak quadriceps stabilizes his knee by
  leaning forward on the affected side & pressing over lower
  thigh by his Ispilateral hand or fingers.
Gluteus maximus gait(BACKWARD
LURCH)
 Due to weakness in gluteus maximus
 muscle, while the body propels
 forward during midstance
 phase,trunk is lurched posterior
 to effect posterior pelvic and
 shifting the centre of gravity
 towards stance hip.
 Seen in poliomyelities & above knee amputation with
 prosthesis.
Stiff hip gait
 When the hip is ankylosed, it is not possible
  to flex at the hip joint during walking to
  clear the ground in the swing phase.
 Hence the person with stiff hip, lifts
the pelvis on that side and swings the leg with
  the pelvis in circumduction and moves it
  forward.
STAMPING/ATAXIC GAIT:
 It occurs in sensory ataxia in which there is loss of sensation
  in lower extremity due to disease processes in peripheral
  nerves, dorsal roots, dorsal column of spinal cord.
 Due to absence of deep position sense,the patient constantly
  observes placing of his feet.
 Hip is hyperflexed & externally rotated & forefoot is
  dorsiflexed to strike ground with a Stamp.
 Seen in peripheral neuritis &
 brain stem lesion in
 children, tabes dorsalis in
 adults.
  Alderman’s gait:
• Seen in Tuberculosis of spine in lower dorsal and upper
 lumbar vertebra.
• Patient walk with head and chest thrown backward and
 protuberant abdomen and legs thrown wide apart.
  GAIT TRAINING
 AIM:
     To achieve safe, easy, effortless normal gait pattern.
Non ambulatory phase
1. Asses and improve the range of movement
2. Treat contractures
3. Improve the cardio respiratory status
4. Shadow walking
5. Assisted device
Ambulatory phase
1. Support by orthotic & prosthesis
2. Parallel bar walking
3. Encourage reciprocal arm swinging
4. Follow other forms of walking
  ➢ Turning
  ➢ Side walk
  ➢ back walk
  ➢ Squatting
  ➢ Getting up
  ➢ Walking on uneven rough surface
Dr. Kavita Meena